Terry Hinds at University of Kentucky - Research Interests, Publications & Academic Profile

Terry Hinds

Associate Professor

University of Kentucky

United States

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Research Interests

Biochemistry

70%

Clinical Nutrition

20%

Fatty Liver

60%

Oxidative Stress

40%

Metabolic Biology

40%

Glucose Metabolism

30%

Nafld

30%

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Recent Grants

Grant: Close

Novel Liver Signaling Pathways Controlling Adiposity

Open Date: 2020-07-01

Close Date: 2025-06-01

Grant: Close

Antioxidant-PPARalpha interaction and hypertension

Open Date: 2014-11-15

Close Date: 2019-10-31

Grant: Close

Fatty acid Control Obesity and the Metabolic Syndrome via TPR proteins

Open Date: 2009-04-13

Close Date: 2011-04-12

Articles16

Impacts of xylazine on fentanyl demand, body weight, and acute withdrawal in rats: A comparison to lofexidine

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Year:

2024

Human soluble prorenin receptor expressed in mouse renal collecting duct shows sex-specific effect on cardiorenal function

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Year:

2024

Glucocorticoids, their uses, sexual dimorphisms, and diseases: new concepts, mechanisms, and discoveries

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Year:

2024

FOXS1 is increased in liver fibrosis and regulates TGFβ responsiveness and proliferation pathways in human hepatic stellate cells

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Year:

2024

Glucocorticoid resistance remodels liver lipids and prompts lipogenesis, eicosanoid, and inflammatory pathways

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Year:

2024

Xylazine Suppresses Fentanyl Consumption During Self-Administration and Induces a Unique Sex-Specific Withdrawal Syndrome That Is Not Altered By Naloxone in Rats

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Year:

2024

Bilirubin Nanoparticle Treatment in Obese Mice Inhibits Hepatic Ceramide Production and Remodels Liver Fat Content

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Year:

2023

Loss of hepatic PPARα in mice causes hypertension and cardiovascular disease

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Year:

2023

The physiology of bilirubin: health and disease equilibrium.

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Year:

2023

Powering the Powerhouse: Heme Oxygenase-1 Regulates Mitochondrial Function in Non-Alcoholic Fatty Liver Disease (NAFLD).

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Year:

2023

Bilirubin Levels Are Negatively Correlated with Adiposity in Obese Men and Women, and Its Catabolized Product, Urobilin, Is Positively Associated with Insulin Resistance

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Year:

2023

Suppressing Hepatic UGT1A1 Increases Plasma Bilirubin, Lowers Plasma Urobilin, Reorganizes Kinase Signaling Pathways and Lipid Species and Improves Fatty Liver Disease

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Year:

2023

Antioxidant Therapy Significantly Attenuates Hepatotoxicity following Low Dose Exposure to Microcystin-LR in a Murine Model of Diet-Induced Non-Alcoholic Fatty Liver Disease

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Year:

2022

Editorial: Emerging talents in Frontiers in Pharmacology: Drug metabolism and transport 2022.

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Year:

2022

Zinc fingers and homeoboxes 2 is required for diethylnitrosamine‐induced liver tumor formation in C57BL/6 mice

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Year:

2022

Molecular mechanisms of metabolic associated fatty liver disease (MAFLD): functional analysis of lipid metabolism pathways.

Article Link

Year:

2022

Collaborators9

William Stoops

Professor

University of Kentucky

UNITED STATES

Martha Peterson

Professor

University of Kentucky College of Medicine

UNITED STATES

Robert Helsley

Assistant Professor

University of Kentucky

UNITED STATES

Steven Haller

Associate Professor

University of Toledo Medical Center

UNITED STATES

Analia S. Loria

Associate Professor

University of Kentucky

UNITED STATES

Justin Fortune Creeden

University of Toledo College of Medicine and Life Sciences

UNITED STATES

Brett T. Spear

University of Kentucky College of Medicine

UNITED STATES

Hunter Moseley

Professor

University of Kentucky

UNITED STATES

Alexandre A. da Silva

University of Mississippi Medical Center

UNITED STATES

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