Ruth Broering at Medical Faculty University of Duisburg‐Essen Essen Germany - Research Interests, Publications & Academic Profile

Ruth Broering

University Name

Germany

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Research Interests

Immunology

90%

Virology

100%

Hepatology

90%

Inflammatory Biology

70%

Liver Fibrosis

50%

B Cell Biology

40%

Antiviral Research

30%

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Recent Grants

Grant: Close

Important roles of HBe and HBs antigens in the evasion of endogenous innate immune responses in Hepatitis B Virus infection.

Open Date: 2021-11-01

Close Date: 2024-10-01

Grant: Close

Impact of parenchymal and nonparenchymal liver cells on Hepatitis B virus infection - Virus host interactions determine immunopathology and chronicity

Open Date: 2018-11-01

Close Date: 2022-07-01

Grant: Close

Proteomic and primary cell preparation services – Quantitative and functional analyses of proteome alterations during chronic viral infection (Z03 (A05))

Open Date: 2013-07-01

Close Date: 2017-12-31

Ruth Broering

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Research Interests

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Recent Grants

Articles16

Hepatitis B surface antigen expression impairs endoplasmic reticulum stress-related autophagic flux by decreasing LAMP2

Nuclear translocation of <scp>YAP</scp> drives <scp>BMI</scp>‐associated hepatocarcinogenesis in hepatitis B virus infection

EGF receptor modulates HEV entry in human hepatocytes

Poly(I:C) Induces Distinct Liver Cell Type-Specific Responses in Hepatitis B Virus-Transgenic Mice In Vitro, but Fails to Induce These Signals In Vivo

Tg1.4HBV-s-rec mice, a crossbred hepatitis B virus-transgenic model, develop mild hepatitis.

Janus kinase-inhibition modulates hepatitis E virus infection.

A ribavirin-induced ORF2 single-nucleotide variant produces defective hepatitis E virus particles with immune decoy function.

Human hepatocyte-enriched miRNA-192-3p promotes HBV replication through inhibiting Akt/mTOR signalling by targeting ZNF143 in hepatic cell lines

Interferon Alpha Induces Cellular Autophagy and Modulates Hepatitis B Virus Replication

Antiviral Toll-like Receptor Signaling in Non-Parenchymal Liver Cells Is Restricted to TLR3

Transcriptome-Wide Analysis of Human Liver Reveals Age-Related Differences in the Expression of Select Functional Gene Clusters and Evidence for a PPP1R10-Governed ‘Aging Cascade’

Peripheral blood iNKT cell activation correlates with liver damage during acute hepatitis C.

In Vivo Mouse Models for Hepatitis B Virus Infection and Their Application.

HBeAg induces liver sinusoidal endothelial cell activation to promote intrahepatic CD8 T cell immunity and HBV clearance.

Hippo Pathway Counter-Regulates Innate Immunity in Hepatitis B Virus Infection.

The impact of hepatitis B surface antigen on natural killer cells in patients with chronic hepatitis B virus infection.

Collaborators6

Eike Steinmann

Susanne Klein-Scory

Konstanze F Winklhofer

Jan-Peter Sowa

Robert K. Gieseler

David Durantel

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