Agata Smogorzewska

Associate Professor

Rockefeller University

United States

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Agata Smogorzewska is an Associate Professor at Rockefeller University in the United States. Her research primarily focuses on the molecular mechanisms underlying Fanconi anemia, as well as DNA repair pathways and their implications in cancer. Recent publications highlight her work on the genetic and structural aspects of DNA repair, particularly in relation to chromosomal stability and replication stress.

Articles (11)

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2024

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2023

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2022

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2022

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2021

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2021

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2021

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2020

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2020

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UNITED STATES

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Simona Giunta

Head of Laboratory; Assistant Professor

Sapienza University of Rome

ITALY

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Agata Smogorzewska

Research Interests

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Articles (11)

FANCA c.3624C>T (p.Ser1208=) is a hypomorphic splice variant associated with delayed onset of Fanconi anemia

Temporary HMCES-DNA cross-link prevents permanent DNA damage.

The structure-specific endonuclease complex SLX4–XPF regulates Tus–Ter-induced homologous recombination

Genomic signature of Fanconi anaemia DNA repair pathway deficiency in cancer.

CENP-A chromatin prevents replication stress at centromeres to avoid structural aneuploidy

Treatment of Fanconi Anemia–Associated Head and Neck Cancer: Opportunities to Improve Outcomes

Endogenous formaldehyde destroys blood stem cells

Esophageal cancer as initial presentation of Fanconi anemia in patients with a hypomorphic FANCA variant

Mechanisms of direct replication restart at stressed replisomes

Suppression of non-homologous end joining does not rescue DNA repair defects in Fanconi anemia patient cells

Distinct roles of BRCA2 in replication fork protection in response to hydroxyurea and DNA interstrand cross-links

Collaborators (6)

Moonjung Jung

Ralph Scully

Hyunseok Kang

Francis Lach

Ramanagouda Ramanagoudr-Bhojappa

Simona Giunta

Make your next move.

Agata Smogorzewska

Research Interests

Explore related searches

Contact this professor

About

Articles (11)

FANCA c.3624C&gt;T (p.Ser1208=) is a hypomorphic splice variant associated with delayed onset of Fanconi anemia

Temporary HMCES-DNA cross-link prevents permanent DNA damage.

The structure-specific endonuclease complex SLX4–XPF regulates Tus–Ter-induced homologous recombination

Genomic signature of Fanconi anaemia DNA repair pathway deficiency in cancer.

CENP-A chromatin prevents replication stress at centromeres to avoid structural aneuploidy

Treatment of Fanconi Anemia–Associated Head and Neck Cancer: Opportunities to Improve Outcomes

Endogenous formaldehyde destroys blood stem cells

Esophageal cancer as initial presentation of Fanconi anemia in patients with a hypomorphic FANCA variant

Mechanisms of direct replication restart at stressed replisomes

Suppression of non-homologous end joining does not rescue DNA repair defects in Fanconi anemia patient cells

Distinct roles of BRCA2 in replication fork protection in response to hydroxyurea and DNA interstrand cross-links

Collaborators (6)

Moonjung Jung

Ralph Scully

Hyunseok Kang

Francis Lach

Ramanagouda Ramanagoudr-Bhojappa

Simona Giunta

Make your next move.

FANCA c.3624C>T (p.Ser1208=) is a hypomorphic splice variant associated with delayed onset of Fanconi anemia