Aki Manninen

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Professor of Cell Biology

University of Oulu

Finland

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About

Professor Aki Manninen is a faculty member in the Department of Cell Biology at the University of Oulu, Finland. His research focuses on the roles of extracellular matrix remodeling, TGFβ1 signaling, and cell junction formation in cancer progression, particularly in prostate and breast cancers. He has published several articles exploring these mechanisms, including studies on the modulation of oncogenic signaling and the development of ureteric structures.

Recent Grants

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Characterization of integrin networks regulating epithelial cell differentiation

Open Date: 2010-08-01

Close Date: 2015-07-01

Grant: Close

The regulatory roles of extracellular matrix and lipid rafts in epithelial cell polarity

Open Date: 2007-01-01

Close Date: 2009-12-01

Articles (13)

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2024

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2023

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2023

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2023

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2022

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2022

Collaborators (9)

Paulina Nastaly

Assistant Professor

Medical University of Gdansk

POLAND

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Markku Varjosalo

University of Helsinki

FINLAND

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Tomasz Wenta

assistant professor

University of Gdańsk

POLAND

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Ulrike Engel

Head of Nikon Imaging Center

Heidelberg University

GERMANY

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Helena Vihinen

University of Helsinki

FINLAND

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Eija Jokitalo

University of Helsinki

FINLAND

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Florence Naillat

FINLAND

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Yosuke Senju

Lecturer

Okayama University

JAPAN

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Juha Saarikangas

Associate Professor

University of Helsinki

FINLAND

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Aki Manninen

Research Interests

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About

Recent Grants

Articles (13)

Remodeling of the extracellular matrix by serine proteases as a prerequisite for cancer initiation and progression

Dampened Regulatory Circuitry of TEAD1/ITGA1/ITGA2 Promotes TGFβ1 Signaling to Orchestrate Prostate Cancer Progression

Dampened Regulatory Circuitry of TEAD1/ITGA1/ITGA2 Promotes TGFβ1 Signaling to Orchestrate Prostate Cancer Progression

HIF2α-dependent Dock4/Rac1-signaling regulates formation of adherens junctions and cell polarity in normoxia

HIF2α-dependent Dock4/Rac1-signaling regulates formation of adherens junctions and cell polarity in normoxia

Actin-rich lamellipodia-like protrusions contribute to the integrity of epithelial cell–cell junctions

Temporally and spatially regulated collagen XVIII isoforms are involved in ureteric tree development via the TSP1-like domain.

GATA2 co-opts TGFβ1/SMAD4 oncogenic signaling and inherited variants at 6q22 to modulate prostate cancer progression

Targeting collagen XVIII improves the efficiency of ErbB inhibitors in breast cancer models.

Targeting collagen XVIII improves the efficiency of ErbB inhibitors in breast cancer models

GATA2 co-opts TGFβ1/SMAD4 oncogenic signaling and inherited variants at 6q22 to modulate prostate cancer progression

Regulation of Kinase Signaling Pathways by α6β4-Integrins and Plectin in Prostate Cancer

Disassembly of α6β4-mediated hemidesmosomal adhesions promotes tumorigenesis in PTEN-negative prostate cancer by targeting plectin to focal adhesions

Collaborators (9)

Paulina Nastaly

Markku Varjosalo

Tomasz Wenta

Ulrike Engel

Helena Vihinen

Eija Jokitalo

Florence Naillat

Yosuke Senju

Juha Saarikangas

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