Akito Nakao

助教 / Assistant Professor

Kyoto University

Japan

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About

Akito Nakao is an Assistant Professor at Kyoto University, Japan. His research focuses on the intricate interactions within cellular signaling systems, particularly involving calcium channels and their associations with conditions affecting cognitive function. Recent work includes studies on brain pH alterations in neuropsychiatric disorders and the innovative use of optochemical techniques in oxygen sensing.

Articles (11)

Year:

2024

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2024

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2024

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2024

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2023

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2023

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2023

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2022

Year:

2022

Year:

2022

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2022

Collaborators (9)

Akira MURAKAMI

特定教授 / Program-Specific Professor

Kyoto University

JAPAN

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Hideo Hagihara

Fujita Health University

JAPAN

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Kazunobu Sawamoto

Professor

Nagoya University

JAPAN

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Tsuyoshi Miyakawa

Fujita Health University

JAPAN

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Jun Yamamoto

Assistant Professor

University of Texas Southwestern Medical School

UNITED STATES

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Kunihiro Tsuchida

JAPAN

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Kota Tamada

Kobe University

JAPAN

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Naoya Ieda

Associate Professor

Hokkaido University

JAPAN

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Masato Sawada

Associate professor

Nagoya University Graduate School of Medicine

JAPAN

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Akito Nakao

Research Interests

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About

Articles (11)

Large-scale animal model study uncovers altered brain pH and lactate levels as a transdiagnostic endophenotype of neuropsychiatric disorders involving cognitive impairment

Large-scale animal model study uncovers altered brain pH and lactate levels as a transdiagnostic endophenotype of neuropsychiatric disorders involving cognitive impairment

Universal roles of the TRPA1 channel in oxygen-sensing

Dynamic remodeling of TRPC5 channel–caveolin-1–eNOS protein assembly potentiates the positive feedback interaction between Ca<sup>2+</sup> and NO signals

An Optochemical Oxygen Scavenger Enabling Spatiotemporal Control of Hypoxia

An Optochemical Oxygen Scavenger Enabling Spatiotemporal Control of Hypoxia

An Optochemical Oxygen Scavenger Enabling Spatiotemporal Control of Hypoxia

Multiple types of navigational information are independently encoded in the population activities of the dentate gyrus neurons

Hippocampus-related cognitive disorders develop in the absence of epilepsy and ataxia in the heterozygous Cacna1a mutant mice tottering

Vital but vulnerable: Human TRPV6 is a trade-off of powerful Ca2+ uptake and susceptibility to epithelial barrier dysfunction

Vital but vulnerable: Human TRPV6 is a trade-off of powerful Ca2+ uptake and susceptibility to epithelial barrier dysfunction Comment

Collaborators (9)

Akira MURAKAMI

Hideo Hagihara

Kazunobu Sawamoto

Tsuyoshi Miyakawa

Jun Yamamoto

Kunihiro Tsuchida

Kota Tamada

Naoya Ieda

Masato Sawada

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