Alexander Dietrich

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University of Munich (LMU)

Germany

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About

Professor Alexander Dietrich is a faculty member at LM-University Munich, Germany. His research focuses on the role of transient receptor potential (TRP) channels in lung biology and disease, particularly in areas such as pulmonary endothelial barrier recovery, cytokine production regulation, and alveolar epithelial barrier integrity. Recent publications highlight his work on electrical cell-substrate impedance sensing and the effects of various agents on lung cell differentiation and toxicity.

Recent Grants

Grant: Close

Endothelial and epithelial activation of TRP channels in toxic lung damage

Open Date: 2018-09-01

Close Date: 2021-08-01

Grant: Close

Reactive oxygen species (ROS)-signaling by TRP channels in the lung

Open Date: 2018-07-01

Close Date: 2022-06-01

Grant: Close

Novel Ca2+ influx pathways in pulmonary hypertension and vascular dysfunction

Open Date:

Close Date:

Grant: Close

TRP function and oxygen signalling in the lung

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Close Date:

Articles (15)

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2025

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2024

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2024

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2024

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2024

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2023

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2023

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2022

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2022

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2021

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2021

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2021

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2020

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2020

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2020

Collaborators (2)

Juan E. Camacho Londoño

Heidelberg University

GERMANY

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Fenja Knoepp

Justus Liebig University Giessen

GERMANY

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Alexander Dietrich

Research Interests

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About

Recent Grants

Articles (15)

TRPV2 channels facilitate pulmonary endothelial barrier recovery after ROS-induced permeability

Electrical cell‐substrate impedance sensing (ECIS) in lung biology and disease

TRPM2 channels are essential for regulation of cytokine production in lung interstitial macrophages

TRPV4 Mediates Alveolar Epithelial Barrier Integrity and Induces ADAM10-Driven E-Cadherin Shedding

Use of FRET-Sensor ‘Mermaid’ to Detect Subtle Changes in Membrane Potential of Primary Mouse PASMCs

An Inhibitory Function of TRPA1 Channels in TGF-β1–driven Fibroblast-to-Myofibroblast Differentiation

Is Zn2+ the new Ca2+ for TRPC6 channels in the myocardium?

Transient Receptor Potential (TRP) Channels in Airway Toxicity and Disease: An Update

An ex vivo perfused ventilated murine lung model suggests lack of acute pulmonary toxicity of the potential novel anticancer agent (−)-englerin A

Transcriptional signatures regulated by TRPC1/C4-mediated Background Ca2+ entry after pressure-overload induced cardiac remodelling

Alkylated epidermal creatine kinase as a biomarker for sulfur mustard exposure: comparison to adducts of albumin and DNA in an in vivo rat study

Ca2+ Signaling by TRPV4 Channels in Respiratory Function and Disease

TRPV4 channels are essential for alveolar epithelial barrier function as protection from lung edema.

Radiation-induced lung toxicity - cellular and molecular mechanisms of pathogenesis, management, and literature review.

Store-operated Ca<sup>2+</sup> entry in primary murine lung fibroblasts is independent of classical transient receptor potential (TRPC) channels and contributes to cell migration.

Collaborators (2)

Juan E. Camacho Londoño

Fenja Knoepp

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