Andre Kleinridders

Prof. Dr.

University of Potsdam

Germany

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Prof. Dr. Andre Kleinridders is a faculty member at the University of Potsdam, Germany. His research focuses on the molecular mechanisms of insulin signaling, obesity, and metabolic processes in neuronal cells, as evidenced by his recent publications. He has explored topics such as the regulation of insulin receptors, the effects of high-fat diets on glycosylation processes, and the influence of insulin on emotional behavior.

Articles (16)

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2024

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2024

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2024

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2021

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2021

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2020

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2020

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2020

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2020

Collaborators (9)

Francis Chaouloff

INSERM

FRANCE

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Steffen Lütke

University of Cologne

GERMANY

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Mathieu Di Miceli

Lecturer

University of Worcester

UNITED KINGDOM

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Michaela Rath

University of Potsdam

GERMANY

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Sigmar Stricker

Professor

GERMANY

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Sébastien BULLICH

Max Planck Florida Institute for Neuroscience

UNITED STATES

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Daniela Cota

University of Bordeaux

FRANCE

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Anna P. Kipp

GERMANY

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Sophie Layé

University of Bordeaux

FRANCE

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Andre Kleinridders

Research Interests

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Articles (16)

High-fat diet alters N-glycosylation of PTPRJ in murine liver

PTPRJ is a negative regulator of insulin signaling in neuronal cells, impacting protein biosynthesis, and neurite outgrowth

DEP-1 is a brain insulin receptor phosphatase that prevents the simultaneous activation of counteracting metabolic pathways.

Lactobacillus rhamnosus Sex-Specifically Attenuates Depressive-like Behavior and Mitigates Metabolic Consequences in Obesity

Insulin modulates emotional behavior through a serotonin-dependent mechanism.

Obesity Hinders the Protective Effect of Selenite Supplementation on Insulin Signaling

Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats.

Hemicentin-1 is an essential extracellular matrix component of the dermal–epidermal and myotendinous junctions

Orexin receptors 1 and 2 in serotonergic neurons differentially regulate peripheral glucose metabolism in obesity.

HSP60 reduction protects against diet-induced obesity by modulating energy metabolism in adipose tissue.

Central Acting Hsp10 Regulates Mitochondrial Function, Fatty Acid Metabolism, and Insulin Sensitivity in the Hypothalamus.

Untangling the effect of insulin action on brain mitochondria and metabolism

Cell autonomous requirement of neurofibromin (Nf1) for postnatal muscle hypertrophic growth and metabolic homeostasis

Cell autonomous requirement of neurofibromin (Nf1) for postnatal muscle hypertrophic growth and metabolic homeostasis.

Central nervous pathways of insulin action in the control of metabolism and food intake.

Interplay of Dietary Fatty Acids and Cholesterol Impacts Brain Mitochondria and Insulin Action.

Collaborators (9)

Francis Chaouloff

Steffen Lütke

Mathieu Di Miceli

Michaela Rath

Sigmar Stricker

Sébastien BULLICH

Daniela Cota

Anna P. Kipp

Sophie Layé

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