Andrew Wagner

Assistant Professor

Bowling Green State University

United States

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Andrew Wagner is an Assistant Professor at Bowling Green State University, USA. His research focuses on innate immune responses, fungal infections, and the mechanisms of antifungal immunity, particularly in relation to Candida albicans and neutrophils. Recent publications include studies on burn wound infection models, the role of GATA1-deficient stem cells, and nutrient utilization in neutrophils.

Articles (10)

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2025

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2025

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2025

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2024

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2023

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2022

Candida albicans is a common cause of human mucosal yeast infections, and invasive candidiasis can be fatal. Antifungal medications are limited, but those targeting the pathogen cell wall or plasma membrane have been effective.

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2022

Year:

2022

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2022

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2021

Collaborators (5)

Todd Reynolds

Associate Professor

University of Tennessee

UNITED STATES

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Nicholas Arp

University of Wisconsin-Madison

UNITED STATES

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Brian M Peters

UNITED STATES

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Todd R. Graham

Vanderbilt University

UNITED STATES

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Trevor Hancock

Assistant Professor

University of Tennessee Health Science Center

UNITED STATES

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Andrew Wagner

Research Interests

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About

Articles (10)

Larval zebrafish burn wound infection model reveals conserved innate immune responses against diverse pathogenic fungi

GATA1-deficient human pluripotent stem cells generate neutrophils with improved antifungal immunity that is mediated by the integrin CD18

Isotonic medium treatment limits burn wound microbial colonisation and improves tissue repair

Activation induces shift in nutrient utilization that differentially impacts cell functions in human neutrophils

Caspofungin-induced β(1,3)-glucan exposure in Candida albicans is driven by increased chitin levels

Cek1 regulates ß(1,3)-glucan exposure through calcineurin effectors in Candida albicans

Lipid Transport by <i>Candida albicans</i> Dnf2 Is Required for Hyphal Growth and Virulence

Mucosal Infection with Unmasked Candida albicans Cells Impacts Disease Progression in a Host Niche-Specific Manner

When Is It Appropriate to Take Off the Mask? Signaling Pathways That Regulate ß(1,3)-Glucan Exposure in Candida albicans

Activation of Cph1 causes ß(1,3)-glucan unmasking in Candida albicans and attenuates virulence in mice in a neutrophil-dependent manner

Collaborators (5)

Todd Reynolds

Nicholas Arp

Brian M Peters

Todd R. Graham

Trevor Hancock

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