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Andrew Waters

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Assistant Professor

University of Cincinnati

United States

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Identification of resistance mechanisms to direct KRAS inhibition in pancreatic cancer

Open Date: 2023-06-15

Close Date: 2026-05-31

Articles (19)

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Collaborators (16)

Ralph Francescone

Assistant Scientist/Assistant Professor

Fox Chase Cancer Center

UNITED STATES

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Mariaelena Pierobon

Professor of Applied Molecular Oncology

University of Sheffield

UNITED KINGDOM

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yuesong wu

Michigan State University

UNITED STATES

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Christopher M. Counter

Duke University Medical Center

UNITED STATES

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DYLAN TAATJES

University of Colorado Boulder

UNITED STATES

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Lee M. Graves

Professor

University of North Carolina at Chapel Hill

UNITED STATES

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Timothy Martin

Assistant Professor

University of Virginia

UNITED STATES

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Antje Schaefer

University of North Carolina at Chapel Hill

UNITED STATES

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Timothy Le Cras

Associate Dean for Graduate Education

Cincinnati Children's Hospital Medical Center

UNITED STATES

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Adrienne Hammill

Professor of Clinical Pediatrics

Cincinnati Children's Hospital Medical Center

UNITED STATES

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Adrienne D. Cox

University of North Carolina at Chapel Hill

UNITED STATES

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Nhan Tran

Professor

Translational Genomics Research Institute

UNITED STATES

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Linda Z Penn

University Health Network

CANADA

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Kirsten Bryant

Research Assistant Professor

University of North Carolina

UNITED STATES

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Clint Stalnecker

Research Assistant Professor

University of North Carolina at Chapel Hill

UNITED STATES

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Jennifer Klomp

Assistant Professor

University of Michigan Health System

UNITED STATES

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Andrew Waters

Research Interests

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Recent Grants

Articles (19)

RAS Inhibitor RMC‐7977 Blocks Vascular Overgrowth of NRASQ61R Mutant Endothelial Cells

CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer.

Human Pancreatic Cancer Single-Cell Atlas Reveals Association of CXCL10+ Fibroblasts and Basal Subtype Tumor Cells

Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer

RAS Pathway Mutations and Therapeutics in Vascular Anomalies

Mediator kinase inhibition impedes transcriptional plasticity and prevents resistance to ERK/MAPK-targeted therapy in KRAS-mutant cancers.

Evaluation of KRAS inhibitor-directed therapies for pancreatic cancer treatment

Concurrent inhibition of oncogenic and wild-type RAS-GTP for cancer therapy.

Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer

TEAD Inhibition Overcomes YAP1/TAZ-Driven Primary and Acquired Resistance to KRASG12C Inhibitors

Functional and biological heterogeneity of KRAS<sup>Q61</sup> mutations.

Concurrent Inhibition of ERK and Farnesyltransferase Suppresses the Growth of HRAS Mutant Head and Neck Squamous Cell Carcinoma

Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth.

CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment

Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition.

Validation of Isoform- and Mutation-Specific RAS Antibodies.

Author Correction: Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer

Low-Dose Vertical Inhibition of the RAF-MEK-ERK Cascade Causes Apoptotic Death of KRAS Mutant Cancers.

Atypical KRASG12R Mutant Is Impaired in PI3K Signaling and Macropinocytosis in Pancreatic Cancer

Collaborators (16)

Ralph Francescone

Mariaelena Pierobon

yuesong wu

Christopher M. Counter

DYLAN TAATJES

Lee M. Graves

Timothy Martin

Antje Schaefer

Timothy Le Cras

Adrienne Hammill

Adrienne D. Cox

Nhan Tran

Linda Z Penn

Kirsten Bryant

Clint Stalnecker

Jennifer Klomp

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