Apurva Tadimari Prabhakar

Assistant Professor

Virginia Commonwealth University

United States

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Articles (6)

Human papillomaviruses are pathogens that cause a host of diseases ranging from benign warts to cancers. There are no therapeutics available for combating these diseases that directly target viral proteins or processes; therefore, we must enhance our understanding of HPV life cycles to assist with identifying novel treatments.

Year:

2023

Year:

2022

Year:

2022

Year:

2022

Year:

2021

Year:

2020

Collaborators (2)

Matthew Day

Lecturer in Biochemistry

Queen Mary University of London

UNITED KINGDOM

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Iain M. Morgan

UNITED STATES

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Apurva Tadimari Prabhakar

Research Interests

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Articles (6)

Human Papillomavirus 16 E2 Interaction with TopBP1 Is Required for E2 and Viral Genome Stability during the Viral Life Cycle

Interaction with TopBP1 Is Required for Human Papillomavirus 16 E2 Plasmid Segregation/Retention Function during Mitosis

A Critical Role for p53 during the HPV16 Life Cycle

Human papillomavirus 16 E2 blocks cellular senescence in response to activation of the DNA damage response.

CK2 Phosphorylation of Human Papillomavirus 16 E2 on Serine 23 Promotes Interaction with TopBP1 and Is Critical for E2 Interaction with Mitotic Chromatin and the Viral Life Cycle

Human Papillomavirus 16 E6 and E7 Synergistically Repress Innate Immune Gene Transcription

Collaborators (2)

Matthew Day

Iain M. Morgan

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