Ari Waisman

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Institue Director and full Professor

University Medical Center of the Johannes Gutenberg University Mainz

Germany

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About

Ari Waisman is the Institute Director and a full Professor at the University Medical Center Mainz, Germany. His research focuses on immune responses related to neurodevelopmental disorders, blood-brain barrier models, and protective signaling in colitis. Recent articles highlight his work on the roles of innate lymphoid cells, microglia, and dendritic cells in various disease models.

Recent Grants

Grant: Close

Era-Net Neuron: MEchanisms of Lymphocytes TRansmigration Across the Blood Brain Barrier

Open Date: 2015-07-01

Close Date: 2018-06-01

Grant: Close

Der Einfluss der Darmflora auf enzephalitogene T-Zellen und die Rolle von IL-17 und IFNgamma in autoimmunen Erkrankungen des ZNS

Open Date: 2015-01-01

Close Date: 2017-06-01

Grant: Close

Regulation of Th17 cells in CNS inflammatory diseases

Open Date: 2014-01-01

Close Date: 2016-12-01

Grant: Close

Regulation of Th17 cells in CNS inflammatory diseases

Open Date: 2014-01-01

Close Date: 2016-12-01

Grant: Close

Role and function of microglia cells in CNS inflammation

Open Date: 2013-05-01

Close Date: 2016-04-01

Articles (17)

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2025

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2022

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2021

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2020

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2020

Collaborators (10)

Eric Vivier

Chief Science Officer

FRANCE

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Björn CLAUSEN

PI/Professor

Johannes Gutenberg Universität Universitätsmedizin

GERMANY

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Patrick Lacolley

Université de Lorraine

FRANCE

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LAIBAIK PARK

Associate Professor of Research

Weill Cornell Medicine

UNITED STATES

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Yassin Elfaki

Senior scientist

AstraZeneca UK Limited

UNITED KINGDOM

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Huige Li

Professor

Johannes Gutenberg Universität Universitätsmedizin

GERMANY

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Susanne Karbach

Universitätsmedizin Mainz

GERMANY

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Ning Xia

Johannes Gutenberg Universität Universitätsmedizin

GERMANY

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Thomas Korn

GERMANY

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David Brea Lopez

Científico titular (Assistant Professor)

Consejo Superior de Investigaciones Cientificas

SPAIN

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Ari Waisman

Research Interests

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About

Recent Grants

Articles (17)

Innate lymphoid cell (ILC) 1- and NK cell-derived, early IFNγ release depends on ICER and promotes protection against Leishmania major infection.

Recent advances in blood-brain barrier-on-a-chip models

Embryo-restricted responses to maternal IL-17A promote neurodevelopmental disorders in mouse offspring

Role of CYLD in brain physiology and pathology

Microglia’s heroic battle against neurodegeneration

Meningeal interleukin-17-producing T cells mediate cognitive impairment in a mouse model of salt-sensitive hypertension

YOD1 sustains NOD2-mediated protective signaling in colitis by stabilizing RIPK2

Exclusive Expression of MyD88 on Dendritic Cells Is Sufficient to Induce Protection against Experimental Leishmaniasis

Hepatocyte Bcl-3 protects from death-receptor mediated apoptosis and subsequent acute liver failure

Correction to: Cross‑reactive, natural IgG recognizing L. major promote parasite internalization by dendritic cells and promote protective immunity

Cross-reactive, natural IgG recognizing L. major promote parasite internalization by dendritic cells and promote protective immunity

B Lymphocyte-Deficiency in Mice Causes Vascular Dysfunction by Inducing Neutrophilia

Repositioning TH cell polarization from single cytokines to complex help

Recirculating IL-1R2+ Tregs fine-tune intrathymic Treg development under inflammatory conditions

Laquinimod dampens IL-1β signaling and Th17-polarizing capacity of monocytes in patients with MS

PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation

Time to activin on pathogenic T cells

Collaborators (10)

Eric Vivier

Björn CLAUSEN

Patrick Lacolley

LAIBAIK PARK

Yassin Elfaki

Huige Li

Susanne Karbach

Ning Xia

Thomas Korn

David Brea Lopez

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