Atsu Aiba

Professor

University of Tokyo

Japan

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About

Professor Atsu Aiba is a faculty member at The University of Tokyo, Japan. His research focuses on various aspects of molecular biology and genetics, with recent publications addressing topics such as fragile X syndrome, circadian rhythms, and the genetic basis of psychiatric disorders. He is also involved in advancements in genome engineering techniques using model organisms.

Articles (13)

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2025

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2024

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2024

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2023

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2021

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2020

Collaborators (15)

Yusuke Kishi

Associate Professor

University of Tokyo

JAPAN

Atsu Aiba

Research Interests

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Articles (13)

FMR1 mutant marmosets show fragile X syndrome phenotypes

Phosphorylation of DNA-binding domains of CLOCK–BMAL1 complex for PER-dependent inhibition in circadian clock of mammalian cells

TRAF7 determines circadian period through ubiquitination and degradation of DBP

Coevolution of the ileum with Brk/Ptk6 family kinases confers robustness to ileal homeostasis

Regulation of De Novo Lipid Synthesis by the Small GTPase Rac1 in the Adipogenic Differentiation of Progenitor Cells from Mouse White Adipose Tissue

mGluR5 Is Substitutable for mGluR1 in Cerebellar Purkinje Cells for Motor Coordination, Developmental Synapse Elimination, and Motor Learning

Rhythmic transcription of Bmal1 stabilizes the circadian timekeeping system in mammals

Loss of calsyntenin paralogs disrupts interneuron stability and mouse behavior

Efficient marmoset genome engineering by autologous embryo transfer and CRISPR/Cas9 technology

Two novel mouse models mimicking minor deletions in 22q11.2 deletion syndrome revealed the contribution of each deleted region to psychiatric disorders

Atrophy of White Adipose Tissue Accompanied with Decreased Insulin-Stimulated Glucose Uptake in Mice Lacking the Small GTPase Rac1 Specifically in Adipocytes

Telencephalon‐specific Alkbh1 conditional knockout mice display hippocampal atrophy and impaired learning

Setd1a Insufficiency in Mice Attenuates Excitatory Synaptic Function and Recapitulates Schizophrenia-Related Behavioral Abnormalities.

Collaborators (15)

Yusuke Kishi

Yuri Shibata

Jun-ichiro Inoue

Dae Wook Kim

Yasushi Kishimoto

Masanobu Kano

Harumi Nakao

Hiroko Kozuka-Hata

Atsushi Kanemitsu

Masanori Hatakeyama

Masaaki Oyama

Yoshitaka Fukada

Eui Min Jeong

和貴 中尾

Jae Kyoung Kim

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