Barbara Sherry

Professor

Feinstein Institute for Medical Research

United States

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Barbara Sherry is a Professor at the Feinstein Institute for Medical Research in the United States. Her research primarily focuses on immunology, chronic diseases, and the molecular mechanisms involved in sepsis and autoimmune conditions. Recent publications include studies on biomarkers in chronic venous ulcerations, the role of T cells in chronic lymphocytic leukemia, and the effects of vagus nerve stimulation on immune responses.

Articles (15)

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2025

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2021

Collaborators (12)

Stefano Vergani

Lund University

SWEDEN

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Myoungsun Son

Assistant Professor

Donald & Barbara Zucker School of Medicine at Hofstra/Northwell

UNITED STATES

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Umair Ahmed

Feinstein Institute for Medical Research

UNITED STATES

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Betsy Barnes

Professor

Hofstra University North Shore Long Island Jewish School of Medicine

UNITED STATES

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Andrea Nicola Mazzarello

Assistant Professor

University of Genoa

ITALY

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Valentin Pavlov

Professor

Northwell Health Feinstein Institutes for Medical Research

UNITED STATES

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Betty Diamond

Institute Head

Feinstein Institute for Medical Research

UNITED STATES

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Stavros Zanos

Associate Professor

Northwell Health Feinstein Institutes for Medical Research

UNITED STATES

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Kostas Stamatopoulos

Visiting Professor

Karolinska Institutet

SWEDEN

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Steven Allen

Associate Chief, Division Of Hematology

UNITED STATES

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Bruce Volpe

Professor

Feinstein Institute for Medical Research

UNITED STATES

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Anita Ng

Feinstein Institute for Medical Research

UNITED STATES

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Barbara Sherry

Research Interests

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Articles (15)

Elevation of Plasma IL-15 and RANTES as Potential Biomarkers of Healing in Chronic Venous Ulcerations: A Pilot Study

CLL crosstalk with naïve T cells enhances the differentiation of IL-22-producing T cells and CLL -cell survival.

Enhanced resistance to Listeria infection in mice surviving sepsis: the role of lipid metabolism and myeloid cell reprogramming.

Vagus nerve stimulation modulates distinct acetylcholine receptors on B cells and limits the germinal center response

Single cell RNA-seq reveals cellular and transcriptional heterogeneity in the splenic CD11b+Ly6Chigh monocyte population expanded in sepsis-surviving mice

Host factor TIMP1 sustains long-lasting myeloid-biased hematopoiesis after severe infection.

Skewing Towards Effector Memory Cells in CLL Is Associated with IGHV -Mutation Status and IFNγ and IL-4

Circulating Th17 T cells at treatment onset predict autoimmune toxicity of PI3Kδ inhibitors.

Galantamine attenuates autoinflammation in a mouse model of familial mediterranean fever.

Thiocarbazate building blocks enable the construction of azapeptides for rapid development of therapeutic candidates

Activated CLL cells regulate IL-17F–producing Th17 cells in miR155-dependent and outcome-specific manners

Identification of Three Unique Clusters of Serum Proteins with Distinctive Functionalities in IGHV-Mutated MBL

Follicular dendritic cell dysfunction contributes to impaired antigen-specific humoral responses in sepsis-surviving mice

Myeloid-derived suppressor cell subtypes differentially influence T-cell function, T-helper subset differentiation, and clinical course in CLL.

Chronic lymphocytic leukemia-like monoclonal B-cell lymphocytosis exhibits an increased inflammatory signature that is reduced in early-stage chronic lymphocytic leukemia.

Collaborators (12)

Stefano Vergani

Myoungsun Son

Umair Ahmed

Betsy Barnes

Andrea Nicola Mazzarello

Valentin Pavlov

Betty Diamond

Stavros Zanos

Kostas Stamatopoulos

Steven Allen

Bruce Volpe

Anita Ng

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