Beate Hagl

University of Augsburg

Germany

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Articles (5)

Year:

2025

Year:

2021

Year:

2021

Year:

2020

Year:

2020

Collaborators (3)

Roland Kappler

LMU University Hospital

GERMANY

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Ejona Rusha

Head of Core Facility

Helmholtz Zentrum München

GERMANY

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Elisabeth Graf

Klinikum rechts der Isar der Technischen Universität München

GERMANY

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Beate Hagl

Research Interests

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Articles (5)

Signal Transducer and Activator of Transcription 3 (STAT3) Variant p.K709N Causes Hyper‐IgE Syndrome Likely by Impaired STAT3‐Dimer Formation

Rescue of STAT3 Function in Hyper-IgE Syndrome Using Adenine Base Editing.

Class Switch Recombination Defects: impact on B cell maturation and antibody responses

Downregulation of SFRP1 is a protumorigenic event in hepatoblastoma and correlates with beta-catenin mutations.

Retained primary teeth in STAT3 hyper-IgE syndrome: early intervention in childhood is essential

Collaborators (3)

Roland Kappler

Ejona Rusha

Elisabeth Graf

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