Beate Sodeik

Professor

Hannover Medical School

Germany

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Professor Beate Sodeik is a faculty member at Hannover Medical School, Germany. Her research primarily focuses on herpes simplex virus pathogenesis, including its assembly, spread, and the interaction with host cell factors. Recent articles authored by Professor Sodeik explore topics such as the role of antimicrobial proteins in viral restriction and the dynamics of viral infection in human primary keratinocytes.

Articles (21)

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2025

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2024

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2021

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2021

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2021

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2020

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2020

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2020

Collaborators (13)

Alberto Rastrojo

Universidad Autónoma de Madrid

SPAIN

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Maija Vihinen‐Ranta

FINLAND

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Christine Goffinet

Associate Professor

Charité - Universitätsmedizin Berlin

GERMANY

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Manutea Christophe Serrero

Aarhus University

DENMARK

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Markus W. Löffler

University Hospital Tübingen

GERMANY

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Antje Mohs

Medizinische Fakultät der RWTH Aachen

GERMANY

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Mathias Hornef

RWTH Aachen University

GERMANY

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Gunther Hartmann

GERMANY

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Peter O'Hare

Imperial College London

UNITED KINGDOM

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Jürgen Harder

GERMANY

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Felix Flomm

Leibniz-Institut für Experimentelle Virologie

GERMANY

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Khaled R. Alkharsah

SAUDI ARABIA

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Christian Wandall Thorball

Lausanne University Hospital (CHUV)

SWITZERLAND

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Beate Sodeik

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Articles (21)

Author Response to Poto et al. (2025): The Intriguing Role of Short‐Form TSLP by Zeitvogel et al.

Collagen XXIII (COL23A1): A novel risk factor for eczema herpeticum.

Herpes simplex virus assembly and spread in murine skin after infection from the outside.

Short‐form thymic stromal lymphopoietin (sfTSLP) restricts herpes simplex virus infection of human primary keratinocytes

Herpes simplex virus type 1 modifies the protein composition of extracellular vesicles to promote neurite outgrowth and neuroinfection

MX2 forms nucleoporin-comprising cytoplasmic biomolecular condensates that lure viral capsids.

The antimicrobial protein RNase 7 directly restricts herpes simplex virus infection of human keratinocytes

A Hitchhiker's Guide Through the Cell: The World According to the Capsids of Alphaherpesviruses.

The role of nuclear pores and importins for herpes simplex virus infection.

A Baseline Cellular Antiviral State Is Maintained by cGAS and Its Most Frequent Naturally Occurring Variant rs610913

The interferon-inducible GTPase MxB promotes capsid disassembly and genome release of herpesviruses

pUL36 Deubiquitinase Activity Augments Both the Initiation and the Progression of Lytic Herpes Simplex Virus Infection in IFN-Primed Cells

Infection-induced chromatin modifications facilitate translocation of herpes simplex virus capsids to the inner nuclear membrane

Assembly of infectious Kaposi’s sarcoma-associated herpesvirus progeny requires formation of a pORF19 pentamer

The journey of herpesvirus capsids and genomes to the host cell nucleus

Free human DNA attenuates the activity of antimicrobial peptides in atopic dermatitis

Human IFITM3 restricts chikungunya virus and Mayaro virus infection and is susceptible to virus-mediated counteraction

Disturbed gut microbiota and bile homeostasis in Giardia-infected mice contributes to metabolic dysregulation and growth impairment

Herpes Simplex Virus 2 Counteracts Neurite Outgrowth Repulsion during Infection in a Nerve Growth Factor-Dependent Manner.

Herpes simplex virus 2 counteracts neurite outgrowth repulsion during infection in a nerve growth factor-dependent manner

The Proteome and Secretome of Cortical Brain Cells Infected With Herpes Simplex Virus.

Collaborators (13)

Alberto Rastrojo

Maija Vihinen‐Ranta

Christine Goffinet

Manutea Christophe Serrero

Markus W. Löffler

Antje Mohs

Mathias Hornef

Gunther Hartmann

Peter O'Hare

Jürgen Harder

Felix Flomm

Khaled R. Alkharsah

Christian Wandall Thorball

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