Bruce R. Levin

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Professor

Emory University

United States

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About

Professor Bruce R. Levin is a faculty member at Emory University in the United States. His research primarily focuses on the interactions between innate immunity, antibiotics, and bacteriophages in bacterial infections. He has published extensively on topics including the evolution of virulence, mutation rates in Staphylococcus aureus, and the dynamics of heteroresistance in Escherichia coli. His recent work also explores the ecological impacts of antimicrobial resistance and bacterial nutrition in the context of antibiotic pharmacodynamics.

Recent Grants

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Population biology and evolution of antibiotic treatment and resistance

Open Date: 2014-09-01

Close Date: 2018-08-01

Articles (15)

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2020

Collaborators (10)

Nic Vega

Assistant Professor

Emory University

UNITED STATES

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Fernando Baquero

Research Professor

Instituto Ramón y Cajal de Investigación Sanitaria

SPAIN

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Ricardo León-Sampedro

ETH Zürich

SWITZERLAND

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Jerónimo Rodríguez-Beltrán

SPAIN

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David Weiss

Associate Professor

Emory University School of Medicine

UNITED STATES

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Roland Regoes

ETH Zürich

SWITZERLAND

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Marie‐Agnès Petit

FRANCE

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Dan I. Andersson

Uppsala University

SWEDEN

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Jacob Choby

Assistant Professor

University of North Carolina at Chapel Hill

UNITED STATES

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Diarmaid Hughes

Professor of Medical Molecular Bacteriology

Uppsala Universitet Medicinska fakulteten

SWEDEN

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Bruce R. Levin

Research Interests

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About

Recent Grants

Articles (15)

The role of innate immunity, antibiotics, and bacteriophages in the course of bacterial infections and their treatment

The tradeoffs between persistence and mutation rates at sub-inhibitory antibiotic concentrations in Staphylococcus aureus

Theoretical considerations and empirical predictions of the pharmaco- and population dynamics of heteroresistance

Short-sighted evolution of virulence for invasive gut microbes: From hypothesis to tests

The evolution of heteroresistance via small colony variants in Escherichia coli following long term exposure to bacteriostatic antibiotics

The contribution of abortive infection to preventing populations of Lactococcus lactis from succumbing to infections with bacteriophage

Author Correction: Bacterial cGAS senses a viral RNA to initiate immunity

What’s the Matter with MICs: Bacterial Nutrition, Limiting Resources, and Antibiotic Pharmacodynamics

The ecological consequences and evolution of retron-mediated suicide as a way to protect Escherichia coli from being killed by phage

The book of Lambda does not tell us that naturally occurring lysogens of Escherichia coli are likely to be resistant as well as immune

Bacterial cGAS senses a viral RNA to initiate immunity

Translational demand is not a major source of plasmid-associated fitness costs

Evaluating the potential efficacy and limitations of a phage for joint antibiotic and phage therapy of Staphylococcus aureus infections

Proximate and ultimate causes of the bactericidal action of antibiotics

It is unclear how important CRISPR-Cas systems are for protecting natural populations of bacteria against infections by mobile genetic elements

Collaborators (10)

Nic Vega

Fernando Baquero

Ricardo León-Sampedro

Jerónimo Rodríguez-Beltrán

David Weiss

Roland Regoes

Marie‐Agnès Petit

Dan I. Andersson

Jacob Choby

Diarmaid Hughes

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