Camilla Ferreira Wenceslau

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Associate Professor

Medical University of South Carolina

United States

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Camilla Ferreira Wenceslau is an Associate Professor at the University of South Carolina School of Medicine, USA. Her research areas include cardiovascular physiology and disease, mechanotransduction, and the molecular mechanisms underlying vascular dysfunction and heart failure. Recent articles authored by Professor Wenceslau explore topics such as endothelial cell plasticity, the effects of adipose tissue signaling, and various aspects of hypertension. She is dedicated to advancing the understanding of cardiovascular health and its associated complications.

Recent Grants

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Intrarenal Arteries Sense Trauma-derived Mitochondrial N-formyl Peptides Leading to Kidney Injury in SIRS

Open Date: 2016-07-01

Close Date: 2021-09-01

Articles (18)

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2021

Collaborators (4)

Cameron McCarthy

Assistant Professor

University of South Carolina

UNITED STATES

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Satoru Eguchi

Lewis Katz School of Medicine at Temple University

UNITED STATES

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keiichi torimoto

University of Occupational and Environmental Health Japan

JAPAN

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Nancy Kanagy

Professor

University of New Mexico School of Medicine

UNITED STATES

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Camilla Ferreira Wenceslau

Research Interests

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About

Recent Grants

Articles (18)

Hypertensive BPH/2J mice exhibit sex-specific heart failure progression and late-life microvascular rarefaction

Orphan Under Pressure: GPR146 as a Mechanotransduction Modulator

Presence of dysfunctional soluble guanylate cyclase in mesenteric resistance arteries from rats with mild ligature-induced periodontitis

Tetrahydrocurcumin Outperforms Curcumin in Preventing Oxidative Stress-Induced Dysfunction in Tert-Butyl Hydroperoxide-Stimulated Cardiac Fibroblasts

Endothelial Cell Phenotypic Plasticity in Cardiovascular Physiology and Disease: Mechanisms and Therapeutic Prospects

Overexpression of adipose tissue ERα enhances PVAT anticontractility via NOX4-derived H2O2 and is protective against high-fat diet-induced dysfunction

Vascular dysfunction occurs prior to the onset of amyloid pathology and Aβ plaque deposits colocalize with endothelial cells in the hippocampus of female APPswe/PSEN1dE9 mice

Milestone Papers on Signal Transduction Mechanisms of Hypertension and Its Complications

Specialized Pro-resolving Mediator Improves Vascular Relaxation via Formyl Peptide Receptor-2

Soluble and insoluble protein aggregates, endoplasmic reticulum stress, and vascular dysfunction in Alzheimer’s disease and cardiovascular diseases

To err, KIR2 that is, on the side of vasodilation in aging

The O-GlcNAc dichotomy: when does adaptation become pathological?

Reply to Boedtkjer and Aalkjaer

Reply to De Mey et al.

Low-dose 1,3-butanediol reverses age-associated vascular dysfunction independent of ketone body β-hydroxybutyrate

Guidelines for the measurement of vascular function and structure in isolated arteries and veins

Ethanol: striking the cardiovascular system by harming the gut microbiota

Opioids Cause Sex-Specific Vascular Changes via Cofilin-Extracellular Signal-Regulated Kinase Signaling: Female Mice Present Higher Risk of Developing Morphine-Induced Vascular Dysfunction than Male Mice

Collaborators (4)

Cameron McCarthy

Satoru Eguchi

keiichi torimoto

Nancy Kanagy

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