Carlo Mischiati

Associate Professor

University of Ferrara

Italy

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About

Dr. Carlo Mischiati is an Associate Professor at the University of Ferrara, Italy. His research encompasses the biological implications of phytochemicals in cancer treatment, the therapeutic potential of biodynamic supplements, and the effects of capsaicin on cellular mechanisms. Recent publications highlight his work on cytokine secretion in liver cells, transglutaminase type 2 in nervous system tumors, and the cytotoxic effects of novel curcumin derivatives on various cancer cell lines.

Articles (14)

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2025

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2024

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2024

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2024

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2023

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2023

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2023

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2023

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2023

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2023

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2022

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2022

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2021

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2021

Collaborators (3)

anna sansone

Institute of Organic Chemistry and Biochemistry

ITALY

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Marta Di Martile

Istituto Regina Elena

ITALY

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Simone Beninati

Associate Professor

Università degli Studi di Roma La Sapienza

ITALY

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Carlo Mischiati

Research Interests

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Articles (14)

Capsaicin and Its Combination with Oleic Acid Affect Membrane Fatty Acid Remodeling and Cytokine–Chemokine Secretion in HepG2 Cells

Therapeutic Potential of a Biodynamic Supplement on Skin Pressure Ulcers: A Randomized Clinical Study

Biological Implications and Functional Significance of Transglutaminase Type 2 in Nervous System Tumors

α-Pinene, a Main Component of Pinus Essential Oils, Enhances the Expression of Insulin-Sensitive Glucose Transporter Type 4 in Murine Skeletal Muscle Cells

Phytochemicals as Immunomodulatory Agents in Melanoma

Characterization of Stable Pyrazole Derivatives of Curcumin with Improved Cytotoxicity on Osteosarcoma Cell Lines

Characterization of Stable Pyrazole Derivatives of Curcumin with Improved Cytotoxicity on Osteosarcoma Cell Lines

Phytochemicals as Immunomodulatory Agents in Melanoma

Cytotoxicity of Isoxazole Curcumin Analogs on Chronic Myeloid Leukemia-Derived K562 Cell Lines Sensitive and Resistant to Imatinib

Cytotoxicity of Isoxazole Curcumin Analogs on Chronic Myeloid Leukemia-Derived K562 Cell Lines Sensitive and Resistant to Imatinib

A Central Contribution of TG2 Activity to the Antiproliferative and Pro-Apoptotic Effects of Caffeic Acid in K562 Cells of Human Chronic Myeloid Leukemia

A Central Contribution of TG2 Activity to the Antiproliferative and Pro-Apoptotic Effects of Caffeic Acid in K562 Cells of Human Chronic Myeloid Leukemia

Reduction in Fatigue Symptoms Following the Administration of Nutritional Supplements in Patients with Multiple Sclerosis

Reduction in Fatigue Symptoms Following the Administration of Nutritional Supplements in Patients with Multiple Sclerosis

Collaborators (3)

anna sansone

Marta Di Martile

Simone Beninati

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