Chiara FIORENTINI

University of Brescia

Italy

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Articles (11)

Year:

2025

Year:

2024

Year:

2024

Year:

2024

Year:

2023

Year:

2023

Year:

2022

Year:

2022

Year:

2022

Year:

2021

Year:

2020

Collaborators (5)

Alessandro BARBON

Full Professor

University of Brescia

ITALY

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Marco Carli

University of Pisa

ITALY

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Alice Filippini

University of Brescia

ITALY

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Elisa Rossini

Collaboratore Professionale di Ricerca Sanitaria

Istituto Zooprofilattico Sperimentale della Lombardia e dell'Emilia Romagna "Bruno Ubertini"

ITALY

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Adriano Angelucci

ITALY

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Chiara FIORENTINI

Research Interests

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Articles (11)

LRRK2-Mediated Neuroinflammation-Induced Neuronal Dysfunctions in a Parkinson’s and Alzheimer’s Disease Cellular Model

Generation of human induced pluripotent stem cell lines derived from three Noonan syndrome patients from a single family carrying the heterozygous PTPN11 c.188 A > G (p.Y63C) mutation

17-β-estradiol potentiates the neurotrophic and neuroprotective effects mediated by the dopamine D3/acetylcholine nicotinic receptor heteromer in dopaminergic neurons

In-vitro Approaches to Investigate the Detrimental Effect of Light on Dopaminergic Neurons

G Protein-Dependent Activation of the PKA-Erk1/2 Pathway by the Striatal Dopamine D1/D3 Receptor Heteromer Involves Beta-Arrestin and the Tyrosine Phosphatase Shp-2

Central nervous system interaction and crosstalk between nAChRs and other ionotropic and metabotropic neurotransmitter receptors

Structural Plasticity of Dopaminergic Neurons Requires the Activation of the D3R-nAChR Heteromer and the PI3K-ERK1/2/Akt-Induced Expression of c-Fos and p70S6K Signaling Pathway

Induced pluripotent stem cells for defining Parkinsonian patient subtypes: a further step toward precision medicine

Ribociclib Cytotoxicity Alone or Combined With Progesterone and/or Mitotane in in Vitro Adrenocortical Carcinoma Cells

Establishment and characterization of induced pluripotent stem cell (iPSCs) line UNIBSi014-A from a healthy female donor

Extracellular clusterin limits the uptake of α‐synuclein fibrils by murine and human astrocytes

Collaborators (5)

Alessandro BARBON

Marco Carli

Alice Filippini

Elisa Rossini

Adriano Angelucci

Make your next move.

Chiara FIORENTINI

Research Interests

Explore related searches

Contact this professor

Articles (11)

LRRK2-Mediated Neuroinflammation-Induced Neuronal Dysfunctions in a Parkinson’s and Alzheimer’s Disease Cellular Model

Generation of human induced pluripotent stem cell lines derived from three Noonan syndrome patients from a single family carrying the heterozygous PTPN11 c.188 A &gt; G (p.Y63C) mutation

17-β-estradiol potentiates the neurotrophic and neuroprotective effects mediated by the dopamine D3/acetylcholine nicotinic receptor heteromer in dopaminergic neurons

In-vitro Approaches to Investigate the Detrimental Effect of Light on Dopaminergic Neurons

G Protein-Dependent Activation of the PKA-Erk1/2 Pathway by the Striatal Dopamine D1/D3 Receptor Heteromer Involves Beta-Arrestin and the Tyrosine Phosphatase Shp-2

Central nervous system interaction and crosstalk between nAChRs and other ionotropic and metabotropic neurotransmitter receptors

Structural Plasticity of Dopaminergic Neurons Requires the Activation of the D3R-nAChR Heteromer and the PI3K-ERK1/2/Akt-Induced Expression of c-Fos and p70S6K Signaling Pathway

Induced pluripotent stem cells for defining Parkinsonian patient subtypes: a further step toward precision medicine

Ribociclib Cytotoxicity Alone or Combined With Progesterone and/or Mitotane in in Vitro Adrenocortical Carcinoma Cells

Establishment and characterization of induced pluripotent stem cell (iPSCs) line UNIBSi014-A from a healthy female donor

Extracellular clusterin limits the uptake of α‐synuclein fibrils by murine and human astrocytes

Collaborators (5)

Alessandro BARBON

Marco Carli

Alice Filippini

Elisa Rossini

Adriano Angelucci

Make your next move.

Generation of human induced pluripotent stem cell lines derived from three Noonan syndrome patients from a single family carrying the heterozygous PTPN11 c.188 A > G (p.Y63C) mutation