Chiho Suzuki-Minakuchi

Assistant Professor

University of Tokyo

Japan

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Chiho Suzuki-Minakuchi is an Assistant Professor at The University of Tokyo, Japan. His research focuses on microbial genetics and bioremediation, as highlighted by his recent articles on the catabolic plasmid pCAR1 in Pseudomonas putida, the degradation of lignin-derived compounds, and the development of screening devices for marine microbial consortia. His work contributes to the understanding of microbial interactions and the use of bacteria in environmental applications.

Articles (12)

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2025

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2024

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2023

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2022

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2022

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2021

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (4)

Kenji Tomita

University of Tokyo

JAPAN

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Hideaki Nojiri

University of Tokyo

JAPAN

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Keisuke Tomita

Tokyo University of Pharmacy and Life Sciences

JAPAN

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Fumihiro Sassa

Kyushu University

JAPAN

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Chiho Suzuki-Minakuchi

Research Interests

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Articles (12)

DNA-binding Affinity of MvaT Homologs Encoded on the Catabolic Plasmid pCAR1 and the Host Chromosome of Pseudomonas putida KT2440

Transcriptional heterogeneity of catabolic genes on the plasmid pCAR1 causes host-specific carbazole degradation.

Draft Genome Sequence of the Marine-Derived, Anticancer Compound-Producing Bacterium <i>Streptomyces</i> sp. Strain GMY01.

Low-cost gel-filled microwell array device for screening marine microbial consortium

The α- and β-Subunit Boundary at the Stem of the Mushroom-Like α3β3-Type Oxygenase Component of Rieske Non-Heme Iron Oxygenases Is the Rieske-Type Ferredoxin-Binding Site.

Azoxystrobin amine: A novel azoxystrobin degradation product from Bacillus licheniformis strain TAB7

A Toxin-Antitoxin System Confers Stability to the IncP-7 Plasmid pCAR1.

A novel gene cluster is involved in the degradation of lignin-derived monoaromatics in Thermus oshimai JL-2.

Oxygen concentration affects frequency and range of transconjugants for the incompatibility (Inc) P-1 and P-7 plasmids pBP136 and pCAR1.

Effects of environmental factors and coexisting substrates on PAH degradation and transcriptomic responses of the defined bacterial consortium OPK.

A Novel Small RNA on the Pseudomonas putida KT2440 Chromosome Is Involved in the Fitness Cost Imposed by IncP-1 Plasmid RP4

H-NS Family Proteins Drastically Change Their Targets in Response to the Horizontal Transfer of the Catabolic Plasmid pCAR1

Collaborators (4)

Kenji Tomita

Hideaki Nojiri

Keisuke Tomita

Fumihiro Sassa

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