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Ching-Chi Chiu

Associate Professor

Chang Gung University

Taiwan

Research Interests

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Professors in Neuropsychology

Articles (16)

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2026

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2026

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2026

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2026

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2026

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2026

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2026

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2026

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2026

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2026

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2025

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2025

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2024

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2023

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2023

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2023

Collaborators (4)

Chih-Mao Huang

Associate Professor

The University of Hong Kong

HONG KONG

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Chia-Hsiung Cheng

Professor

Chang Gung University

TAIWAN

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Andrea Spitaleri

Researcher

Human Technopole

ITALY

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Ann-Joy Cheng

Professor

Chang Gung University

TAIWAN

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Ching-Chi Chiu

Research Interests

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Articles (16)

Drug repurposing targeting miRNA–mRNA networks to mitigate areca nut-induced head and neck cancer

Altered theta-band sensory gating in individuals with high sensory processing sensitivity (SPS)

Neurophysiological correlates of sensory processing sensitivity: Altered alpha oscillations during auditory change detection

Human iPSC-EV-loaded nanofiber stent coatings accelerate vascular repair by enhancing EGFR/HIF-1α signaling and suppressing ROCK1-mediated remodeling

Resveratrol isomers with opposing activities target endonuclease G to modulate neurodegeneration and mitochondrial elimination

Loss-of-Function (G603R) Lrp10 Fails to Downregulate mRNA of Pathologic α-Synuclein and Causes Neurodegeneration of Substantia Nigra Dopaminergic Cells in Parkinson's Disease Knockin Mice.

Lrp10 insufficiency upregulates mRNA and protein of neurotoxic α-synuclein and causes degeneration of substantia nigra dopaminergic neurons in heterozygous or homozygous Lrp10 knockout mice.

FTDP-17T Mutations Promote Formation of Phosphorylated FTDP-17T TAU Oligomers That Cause Degeneration of Dopaminergic and Hippocampal Neurons via Activating ER Stress and Mitochondrial Pro-apoptotic Cascades.

ALDH2 activation protects against mutant TOMM40-mediated mitochondrial dysfunction and neurodegeneration in Alzheimer's disease

PARK19 truncation mutant Dnajc6 causes lysosomal deficiency-induced upregulation of pathologic α-synuclein and neurodegeneration of substantia nigra dopaminergic cells in PARK19 knockin mice.

ABCA7 variants impact phosphatidylcholine and mitochondria in neurons

Absence of Rab39b-induced macroautophagy impairment increases neurotoxic α-synuclein and causes degeneration of substantia nigra dopaminergic neurons in mouse model of X-linked Parkinson's disease.

Downregulation of Protease Cathepsin D and Upregulation of Pathologic α-Synuclein Mediate Paucity of DNAJC6-Induced Degeneration of Dopaminergic Neurons

HCH6-1, an antagonist of formyl peptide receptor-1, exerts anti-neuroinflammatory and neuroprotective effects in cellular and animal models of Parkinson's disease.

<i>TOMM40</i> Genetic Variants Cause Neuroinflammation in Alzheimer’s Disease

Deficiency of RAB39B Activates ER Stress-Induced Pro-apoptotic Pathway and Causes Mitochondrial Dysfunction and Oxidative Stress in Dopaminergic Neurons by Impairing Autophagy and Upregulating α-Synuclein.

Collaborators (4)

Chih-Mao Huang

Chia-Hsiung Cheng

Andrea Spitaleri

Ann-Joy Cheng

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