Christos Chadjichristos

Associate Professor

INSERM

France

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Christos Chadjichristos is an Associate Professor at INSERM, France. His research focuses on acute kidney injury, kidney fibrosis, and the molecular mechanisms underlying glomerulopathies. Recent publications include studies on the prognostic implications of subclinical AKI in critically ill patients and the role of connexin 43 in renal function. He also investigates biomarkers for cardiorenal injuries and the crosstalk between glomerular endothelial cells and podocytes in renal diseases.

Articles (18)

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2025

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2024

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2022

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2022

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2022

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2021

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2021

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2020

Collaborators (7)

Lefteris Siamantouras

Senior Lecturer Biomedical Engineering

University of Lincoln

UNITED KINGDOM

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Paul Squires

Prof

University of Lincoln

UNITED KINGDOM

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Michael Hultström

Uppsala University

SWEDEN

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Benjamin Glenn Chousterman

Associate-Professor (MCU-PH)

Université Paris-Saclay

FRANCE

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Sahil ADRIOUCH

Professor of Immunology and Biotherapy

Université de Rouen Faculté de Médecine et Pharmacie

FRANCE

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Antoine Roquilly

Prof

University Hospital

FRANCE

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Emmanuel Gautier

INSERM UMR 1236

FRANCE

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Christos Chadjichristos

Research Interests

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Articles (18)

Postoperative subclinical AKI is frequent and is linked to a poor long-term prognosis in critically ill patients

Progression of Kidney Fibrosis after Sepsis

The differential expression of MAGI2 in glomerulopathies and its application as a molecular discriminator of podocytopathies

Endothelial-specific deletion of connexin 43 improves renal function and structure after acute kidney injury

The role of the tricellular junction protein ILDR2 in glomerulopathies: Expression patterns and functional insights

Super‐resolved highly multiplexed immunofluorescence imaging for precise protein localization and podocyte ultrastructure

Galectin-3 and Soluble CD146 Identify Cardiorenal Injuries in Severe Burn Patients: A Biomarker-Based Approach

Crosstalk mechanisms between glomerular endothelial cells and podocytes in renal diseases and kidney transplantation

The role of CD146 in renal disease: from experimental nephropathy to clinics

The Angiogenesis Inhibitor Isthmin-1 (ISM1) Is Overexpressed in Experimental Models of Glomerulopathy and Impairs the Viability of Podocytes

Connexin-43 hemichannels orchestrate NOD-like receptor protein-3 (NLRP3) inflammasome activation and sterile inflammation in tubular injury

Connexin 43: A Target for the Treatment of Inflammation in Secondary Complications of the Kidney and Eye in Diabetes

Deletion of Notch3 Impairs Contractility of Renal Resistance Vessels Due to Deficient Ca<sup>2+</sup> Entry

The Role of Connexin 43 in Renal Disease: Insights from In Vivo Models of Experimental Nephropathy

Galectin-3 in Kidney Diseases: From an Old Protein to a New Therapeutic Target

Super‐resolved local recruitment of CLDN5 to filtration slits implicates a direct relationship with podocyte foot process effacement

Endothelial-Specific Deletion of CD146 Protects Against Experimental Glomerulonephritis in Mice

Blocking Connexin-43 mediated hemichannel activity protects against early tubular injury in experimental chronic kidney disease

Collaborators (7)

Lefteris Siamantouras

Paul Squires

Michael Hultström

Benjamin Glenn Chousterman

Sahil ADRIOUCH

Antoine Roquilly

Emmanuel Gautier

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