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Cynthia A. Reinhart-King

Rice University

United States

Research Interests

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Articles (22)

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2026

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2026

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2026

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2026

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2026

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2026

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2025

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2025

Priming in confined, collagen microtracks increases speed and mitochondrial localization to the front of the cell, even after cells have transitioned out of confinement. Disrupting proper focal adhesion formation decreases this memory.

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2024

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2024

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2024

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2024

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2024

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2024

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2024

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2023

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2023

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2023

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2022

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Assistant Professor

University of Arkansas

UNITED STATES

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Cynthia A. Reinhart-King

Research Interests

Explore related searches

Articles (22)

Transcriptional Heterogeneity Underlying Cancer Cell Migration Converges on Shared Regulatory Programs

Nonmuscle α-Actinin-4 Couples Sarcomere Function to Cardiac Remodeling

Targeting extracellular matrix mechanobiology to suppress solid tumor progression

Mechanobiology of solid tumor vasculature: implications for tumor progression

Cellular mechanometabolism: stimuli and implications

Mechanometabolism of cell adhesion: Vinculin regulates bioenergetics via RhoA-ROCK

Mechanometabolism: recent findings on the intersection of cell adhesion, cell migration, and metabolism

Matrix Stiffness-Mediated DNA Methylation in Endothelial Cells

A "non-muscle" alpha-actinin is an intrinsic component of the cardiac Z-disc and regulates sarcomere turnover, contractility, and heart remodeling

Confinement primes cells for faster migration by polarizing active mitochondria

Cellular Energy Cycle Mediates an Advection‐Like Forward Cell Flow to Support Collective Invasion

Tension directs cancer cell migration over fiber alignment through energy minimization

Matrix stiffness increases energy efficiency of endothelial cells

Collagen density regulates tip–stalk cell rearrangement during angiogenesis via cellular bioenergetics

The movement of mitochondria in breast cancer: internal motility and intercellular transfer of mitochondria

Confinement primes cells for faster migration by polarizing active mitochondria

Mitochondrial transfer from cancer-associated fibroblasts increases migration in aggressive breast cancer

Cellular mechanosignaling for sensing and transducing matrix rigidity

Forces exerted and transduced by cancer‐associated fibroblasts during cancer progression

AGE-breaker ALT711 reverses glycation-mediated cancer cell migration

Cancer cell metabolic plasticity in migration and metastasis

A discrete interface in matrix stiffness creates an oscillatory pattern of endothelial monolayer disruption

Collaborators (6)

Brenton Hoffman

Quinn Wells

Dharmendra Choudhary

Jing Wang

Erdem Tabdanov

Jian Zhang

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