Daniela Kramer

Professor

Johannes Gutenberg Universität Mainz

Germany

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About

Daniela Kramer is a Professor at Johannes Gutenberg University Mainz in Germany. Her research focuses on inflammation, with recent articles addressing psoriasis biomarkers, immune response mechanisms in solid tumors, and the role of reactive oxygen species in inflammatory conditions. She contributes significantly to the understanding of regulatory T-cell differentiation and the immunomodulatory effects of various biochemical factors in skin diseases.

Articles (10)

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2024

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2024

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2024

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2023

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2023

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2023

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2023

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2023

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2021

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2021

Collaborators (8)

Katharina Kommoss

University Hospital Heidelberg

GERMANY

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Bernd Pichler

University of Tuebingen

GERMANY

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Martin Stacey

Associate Professor

University of Leeds

UNITED KINGDOM

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Dominik Sonanini

University of Tübingen

GERMANY

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Wiebke Hansen

University Hospital Essen

GERMANY

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Thomas A. Kufer

GERMANY

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Andre Martins

W3 Full Professor

University of Tübingen

GERMANY

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Caroline Mann

University Medical Center of the Johannes Gutenberg University Mainz

GERMANY

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Daniela Kramer

Research Interests

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Articles (10)

Epidermal proteomics demonstrates Elafin as a psoriasis‐specific biomarker and highlights increased anti‐inflammatory activity around psoriatic plaques

naRNA-LL37 composite DAMPs define sterile NETs as self-propagating drivers of inflammation.

The atypical IκB family member Bcl3 determines differentiation and fate of intestinal RORγt<sup>+</sup> regulatory T-cell subsets.

Acidosis-mediated increase in IFN-γ-induced PD-L1 expression on cancer cells as an immune escape mechanism in solid tumors

A simple tool for evaluation of inflammation in psoriasis: Neutrophil-to-lymphocyte and platelet-to-lymphocyte ratio as markers in psoriasis patients and related murine models of psoriasis-like skin disease.

IL-17A-driven psoriasis is critically dependent on IL-36 signaling.

Immunomodulatory Effects of Histone Variant H2A.J in Ionizing Radiation Dermatitis.

Reactive oxygen species produced by myeloid cells in psoriasis as a potential biofactor contributing to the development of vascular inflammation.

EXPRESSION OF E8^E2 IS REQUIRED FOR WART FORMATION BY MOUSE PAPILLOMAVIRUS 1 IN VIVO.

Immunomodulatory role of reactive oxygen species and nitrogen species during T cell-driven neutrophil-enriched acute and chronic cutaneous delayed-type hypersensitivity reactions.

Collaborators (8)

Katharina Kommoss

Bernd Pichler

Martin Stacey

Dominik Sonanini

Wiebke Hansen

Thomas A. Kufer

Andre Martins

Caroline Mann

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