Daniela POLLAK

Professor for Behavioral Biology

Medical University of Vienna

Austria

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Professor Daniela Pollak is a faculty member at the Medical University of Vienna, Austria. Her research focuses on behavioral biology, with recent articles exploring topics such as the effects of maternal immune activation, feeding behavior modulated by brain circuits, and the impact of polyunsaturated fatty acids on fetal brain development. Additionally, she investigates the role of specific neural pathways in maternal behavior and the behavioral implications of genetic and environmental factors.

Articles (10)

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2024

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2024

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2023

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2022

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2022

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2022

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2021

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2020

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2020

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2020

Collaborators (7)

Peer Wulff

Kiel University

GERMANY

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Spyros Sideromenos

Medical University of Vienna

AUSTRIA

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Francisco Monje

Medical University of Vienna

AUSTRIA

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Wolfgang Winnicki

Medical University of Vienna

AUSTRIA

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Klaus Schmetterer

Medical University of Vienna

AUSTRIA

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Tibor Harkany

Karolinska Institutet

SWEDEN

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Erik Keimpema

AUSTRIA

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Daniela POLLAK

Research Interests

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Articles (10)

Differential effects of purified low molecular weight Poly(I:C) in the maternal immune activation model depend on the laboratory environment

A brainstem–hypothalamus neuronal circuit reduces feeding upon heat exposure

Adverse effects of gestational ω‐3 and ω‐6 polyunsaturated fatty acid imbalance on the programming of fetal brain development

Preclinical Establishment of a Divalent Vaccine against SARS-CoV-2

Uncoupling Protein-1 Modulates Anxiety-Like Behavior in a Temperature-Dependent Manner

An accessory prefrontal cortex–thalamus circuit sculpts maternal behavior in virgin female mice

Secretagogin marks amygdaloid PKCδ interneurons and modulates NMDA receptor availability

STAT3 in the dorsal raphe gates behavioural reactivity and regulates gene networks associated with psychopathology.

VEGF Treatment Ameliorates Depression-Like Behavior in Adult Offspring After Maternal Immune Activation.

Severe hydroxymethylbilane synthase deficiency causes depression-like behavior and mitochondrial dysfunction in a mouse model of homozygous dominant acute intermittent porphyria.

Collaborators (7)

Peer Wulff

Spyros Sideromenos

Francisco Monje

Wolfgang Winnicki

Klaus Schmetterer

Tibor Harkany

Erik Keimpema

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