David A. Jacobson

Professor

Vanderbilt University

United States

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Articles (12)

Year:

2024

Year:

2024

Year:

2022

Year:

2022

Year:

2022

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2021

Year:

2021

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2021

Year:

2021

Year:

2021

Year:

2021

Year:

2021

Collaborators (13)

Anandharajan Rathinasabapathy

Research Assistant Professor

Vanderbilt University Medical Center

UNITED STATES

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Sarah Flanagan

Associate Professor of Genomic Medicine and Sir Henry Dale Fellow

University of Exeter

UNITED KINGDOM

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Aida Martinez-Sanchez

Imperial College London

UNITED KINGDOM

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Thomas Galletta

Assistant Professor of Clinical Pediatrics

Cincinnati Children's Hospital Medical Center

UNITED STATES

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Karin Bosma

Vanderbilt University Medical Center

UNITED STATES

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Taiyi Kuo

Columbia University Irving Medical Center

UNITED STATES

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Kei Sakamoto

Professor

University of Copenhagen

DENMARK

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Darren Tyson

Research Associate Professor

Vanderbilt University School of Medicine

UNITED STATES

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Ling Yan

Vanderbilt University Medical Center

UNITED STATES

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Sarah Graff

Assistant Professor

Lipscomb University

UNITED STATES

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Peggy L. Kendall

UNITED STATES

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Tiziana Sanavia

Assistant Professor

University of Turin

ITALY

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Daniel Segrè

Professor

Boston University

UNITED STATES

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David A. Jacobson

Research Interests

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Articles (12)

Purinergic Ca<sup>2+</sup> Signaling as a Novel Mechanism of Drug Tolerance in BRAF-Mutant Melanoma

Disrupted Endoplasmic Reticulum Ca<sup>2+</sup> Handling: A Harβinger of β-Cell Failure

Liraglutide increases islet Ca<sup>2+</sup> oscillation frequency and insulin secretion by activating hyperpolarization-activated nucleotide-gated channels.

Glucose-Dependent miR-125b is a Negative Regulator of β-Cell Function.

Glucose-6-phosphatase catalytic subunit 2 negatively regulates glucose oxidation and insulin secretion in pancreatic β-cells.

Sex-biased islet β cell dysfunction is caused by the MODY MAFA S64F variant by inducing premature aging and senescence in males

Temporal Transcriptome Analysis Reveals Dynamic Gene Expression Patterns Driving β-Cell Maturation.

KCNK3 Mutation Causes Altered Immune Function in Pulmonary Arterial Hypertension Patients and Mouse Models

Antagonistic epistasis of Hnf4α and FoxO1 metabolic networks through enhancer interactions in β-cell function

A KCNK16 mutation causing TALK-1 gain of function is associated with maturity-onset diabetes of the young.

Channeling β-cell Maturity: KATP Surface Localization Imparts Glucose Sensing.

TRPM7 is a crucial regulator of pancreatic endocrine development and high-fat-diet-induced β-cell proliferation.

Collaborators (13)

Anandharajan Rathinasabapathy

Sarah Flanagan

Aida Martinez-Sanchez

Thomas Galletta

Karin Bosma

Taiyi Kuo

Kei Sakamoto

Darren Tyson

Ling Yan

Sarah Graff

Peggy L. Kendall

Tiziana Sanavia

Daniel Segrè

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