David A. Scott

Associate Dean for Research and Enterprise / Professor

University of Louisville School of Dentistry

United States

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About

David A. Scott is the Associate Dean for Research and Enterprise and a Professor at the University of Louisville School of Dentistry, USA. His research focuses on oral microbiology, with recent articles exploring the pathogenicity of Filifactor alocis, the effects of tobacco on biofilm formation by oral microbes, and the antimicrobial properties of cannabis-derived compounds. His work contributes to a deeper understanding of periodontal pathogens and their interactions with the oral microbiome.

Articles (13)

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2025

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2024

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2024

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2024

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2024

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2022

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2022

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2021

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2021

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2020

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2020

Collaborators (4)

Daniel Miller

Assistant Professor

Virginia Commonwealth University

UNITED STATES

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Fata Moradali

Assistant Professor

University of Louisville School of Dentistry

UNITED STATES

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Michael Sekula

University of Louisville

UNITED STATES

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Shirin Ghods

Assistant Professor

University of Louisville

UNITED STATES

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David A. Scott

Research Interests

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Articles (13)

Filifactor alocis Pathogenicity Requires TLR2 and the Oral Microbiome

Tobacco‐enhanced biofilm formation by Porphyromonas gingivalis and other oral microbes

Synthesis and Characterization of a Sustained Nitric Oxide-Releasing Orthodontic Elastomeric Chain for Antimicrobial Action

The transcriptomic response to cannabidiol of Treponema denticola, a phytocannabinoid‐resistant periodontal pathogen

Delineation of global, absolutely essential and conditionally essential pangenomes of Porphyromonas gingivalis.

Tobacco smoke exacerbates <i>Filifactor alocis</i> pathogenicity

The Antimicrobial Properties of Cannabis and Cannabis-Derived Compounds and Relevance to CB2-Targeted Neurodegenerative Therapeutics

Atypical cyclic di-AMP signaling is essential for Porphyromonas gingivalis growth and regulation of cell envelope homeostasis and virulence.

Potential Mechanisms Underlying Marijuana-Associated Periodontal Tissue Destruction.

The RagA and RagB proteins of Porphyromonas gingivalis.

Porphyromonas gingivalis infection exacerbates oesophageal cancer and promotes resistance to neoadjuvant chemotherapy.

Inherently and Conditionally Essential Protein Catabolism Genes of Porphyromonas gingivalis.

JAK3 restrains inflammatory responses and protects against periodontal disease through Wnt3a signaling.

Collaborators (4)

Daniel Miller

Fata Moradali

Michael Sekula

Shirin Ghods

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