David Basanta

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Professor

Moffitt Cancer Center

United States

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About

David Basanta is a Professor at Moffitt Cancer Center in the United States. His research explores various facets of cancer biology, including tumor evolution, resistance mechanisms, and the interplay between microenvironments and therapy responses. Recent publications highlight his work on agent-based modeling of tumor dynamics and insights into the role of the bone ecosystem in myeloma relapse.

Recent Grants

Grant: Close

Integrated Modeling of TGF-Beta Inhibition in Prostate to Bone Metastases

Open Date: 2012-06-01

Close Date: 2013-05-31

Articles (12)

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2025

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2024

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2020

Collaborators (7)

Daria Miroshnychenko

H Lee Moffitt Cancer Center and Research Institute

UNITED STATES

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Arturo Araujo

University of Roehampton

UNITED KINGDOM

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Shannon Mumenthaler

University of Southern California

UNITED STATES

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Robert Noble

Lecturer

City, University of London

UNITED KINGDOM

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Haralampos Hatzikirou

UNITED ARAB EMIRATES

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Manish Kohli

Professor

Huntsman Cancer Institute

UNITED STATES

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Meghan Ferrall-Fairbanks

Assistant Professor

University of Florida

UNITED STATES

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David Basanta

Research Interests

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About

Recent Grants

Articles (12)

The adaptive state determines the impact of mutations on evolving populations

Dissecting the Spatially Restricted Effects of Microenvironment-Mediated Resistance on Targeted Therapy Responses

A seven‐step guide to spatial, agent‐based modelling of tumour evolution

The bone ecosystem facilitates multiple myeloma relapse and the evolution of heterogeneous drug resistant disease

A survey of open questions in adaptive therapy: Bridging mathematics and clinical translation

Stroma-Mediated Breast Cancer Cell Proliferation Indirectly Drives Chemoresistance by Accelerating Tumor Recovery between Chemotherapy Cycles

Integrated computational and in vivo models reveal Key Insights into macrophage behavior during bone healing

Roadmap on plasticity and epigenetics in cancer

Quantification and Optimization of Standard-of-Care Therapy to Delay the Emergence of Resistant Bone Metastatic Prostate Cancer

Spontaneous cell fusions as a mechanism of parasexual recombination in tumour cell populations

Selection-driven tumor evolution with public goods leads to patterns of clonal expansion consistent with neutral growth

Hybrid Automata Library: A flexible platform for hybrid modeling with real-time visualization

Collaborators (7)

Daria Miroshnychenko

Arturo Araujo

Shannon Mumenthaler

Robert Noble

Haralampos Hatzikirou

Manish Kohli

Meghan Ferrall-Fairbanks

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