David Crossman

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Assistant Professor/Bioinformatics Director

The University of Alabama

United States

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About

David Crossman is an Assistant Professor and Bioinformatics Director at the University of Alabama at Birmingham, USA. His research focuses on bioinformatics, particularly in the context of childhood cancer, immune responses to infections, and the molecular mechanisms underlying various malignancies. Recent publications explore topics such as pathogenic mutations in childhood cancer survivors, immune responses to human cytomegalovirus, and the impact of therapy-related myeloid neoplasms.

Recent Grants

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CLEIDOCRANIAL DYSPLASIA DENTAL PHENOTYPE AND GENOTYPE CORRELATIONS

Open Date: 2011-07-13

Close Date: 2014-06-30

Articles (16)

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Collaborators (17)

Anna Brozyna

Professor and Chair

Nicolaus Copernicus University

POLAND

David Crossman

Research Interests

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About

Recent Grants

Articles (16)

Germline Pathogenic/Likely Pathogenic Mutations and Subsequent Neoplasms Among Childhood Cancer Survivors: A Report From the Children's Oncology Group ALTE03N1 Study

Tissue-Resident CD8+ T Cells as Mediators of Protective Immunity in Breast Milk Transmission of Human Cytomegalovirus

Malignant Melanoma: An Overview, New Perspectives, and Vitamin D Signaling

Sex-Based Differences in Risk of Therapy-Related Myeloid Neoplasms

Clonal Hematopoiesis and Therapy-Related Myeloid Neoplasms After Autologous Transplant for Hodgkin Lymphoma

Metabolic adaptation to tyrosine kinase inhibition in leukemia stem cells

Cytokine storm–based mechanisms for extrapulmonary manifestations of SARS-CoV-2 infection

Low c-Kit expression identifies primitive, therapy-resistant CML stem cells

Impaired PPARγ activation by cadmium exacerbates infection-induced lung injury

Altered Peripheral Blood Gene Expression in Childhood Cancer Survivors With Anthracycline‐Induced Cardiomyopathy – A COG‐ALTE03N1 Report

Intrinsic IL-2 production by effector CD8 T cells affects IL-2 signaling and promotes fate decisions, stemness, and protection

Metabolic Alterations and WNT Signaling Impact Immune Response in HGSOC

Suppression of SIN3A by miR-183 Promotes Breast Cancer Metastasis

Disruption of Endosomal Sorting in Schwann Cells Leads to Defective Myelination and Endosomal Abnormalities Observed in Charcot-Marie-Tooth Disease

Targeting Acid Ceramidase Inhibits Glioblastoma Cell Migration through Decreased AKT Signaling

CRISPR/Cas9-mediated knockout of PIM3 suppresses tumorigenesis and cancer cell stemness in human hepatoblastoma cells

Collaborators (17)

Anna Brozyna

Maria Del Nogal Avila

Tarek Magdy

A. Brent Carter

Allan J. Zajac

Elliot Lefkowitz

Purnima Singh

Tae-Kang Kim

Robert Tuckey

Casey Weaver

Rui Lu

rebecca arend

Steffanie Sabbaj

Shuko Harada

Rasheed Gbadegesin

Zorica Janjetovic

Ravi Bhatia

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