David Davis

Research Assistant Professor

University of Miami School of Medicine

United States

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David Davis is a Research Assistant Professor at the University of Miami School of Medicine in the United States. His research primarily focuses on neurodegenerative diseases, including Alzheimer's disease and Parkinson's disease, as well as the impact of environmental toxins on neurodegeneration. Recent publications highlight his work on transcriptomic analyses related to brain disorders and the role of immune-related gene networks in conditions such as PTSD and schizophrenia.

Articles (14)

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2025

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2021

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2020

Collaborators (8)

Regina Vontell

Assistant Professor

University of Miami Hospital

UNITED STATES

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Vanessa Tan

Macquarie University

AUSTRALIA

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Lecturer

University of Barcelona

SPAIN

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David Davis

Research Interests

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Articles (14)

Alzheimer’s disease signatures in the brain transcriptome of Estuarine Dolphins

Increased inflammasome protein expression identified in microglia from postmortem brains with schizophrenia

TDP-43 and Alzheimer’s Disease Pathology in the Brain of a Harbor Porpoise Exposed to the Cyanobacterial Toxin BMAA

A multi-region single nucleus transcriptomic atlas of Parkinson’s disease

Detection of β-N-methylamino-l-alanine in postmortem olfactory bulbs of Alzheimer's disease patients using UHPLC-MS/MS: An autopsy case-series study.

PTSD, major depression, and advanced transcriptomic age in brain tissue.

Single nucleus multi-omics identifies human cortical cell regulatory genome diversity.

CpH methylome analysis in human cortical neurons identifies novel gene pathways and drug targets for opioid use disorder.

BMAA, Methylmercury, and Mechanisms of Neurodegeneration in Dolphins: A Natural Model of Toxin Exposure

Gene expression in the dorsolateral and ventromedial prefrontal cortices implicates immune-related gene networks in PTSD.

Gene expression correlates of advanced epigenetic age and psychopathology in postmortem cortical tissue.

Is Exposure to BMAA a Risk Factor for Neurodegenerative Diseases? A Response to a Critical Review of the BMAA Hypothesis.

Population-scale tissue transcriptomics maps long non-coding RNAs to complex disease

Transcriptomic organization of the human brain in post-traumatic stress disorder.

Collaborators (8)

Regina Vontell

Vanessa Tan

Juan Pablo de Rivero Vaccari

Rongxin Fang

Karla Kirkegaard

Joshua Akey

Georgios Voloudakis

Diego Garrido-Martín

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