David Engblom

Professor

Linköping University

Sweden

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About

David Engblom is a Professor at Linköping University in Sweden. His research encompasses various topics, including the role of melanocortin receptors in affect and motor control, the effects of inflammation on behavior, and the neurobiological mechanisms underlying febrile responses. Recent publications reflect his interest in neuroinflammation, behavior, and the physiological impacts of resistance training and diet. Engblom's work contributes to a deeper understanding of the intersection between neuroscience and physical health.

Articles (14)

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2025

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2024

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2024

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2024

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2023

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2023

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2022

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2021

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2021

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2021

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2021

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2020

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2020

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2020

Collaborators (5)

Anders Blomqvist

Professor

Linköping University

SWEDEN

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Sigrid Nilsson

Linköping University

SWEDEN

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Anna Mathia Klawonn

Associate professor, group leader

Aarhus University

DENMARK

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Michael Fritz

Professor

GERMANY

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Emilia Berin

Linköping University

SWEDEN

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David Engblom

Research Interests

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About

Articles (14)

Melanocortin 4 receptor-expressing neurons in the lateral stripe of the striatum regulate affect and motor control

NK1 receptor mediates cerebral cellular and extracellular morphological changes during the LPS-induced febrile response

Social transmission of inflammation in mice

Depression: Monocytes on my mind

Melanocortin-4 receptors on neurons in the parabrachial nucleus mediate inflammation-induced suppression of food-seeking behavior.

ACETAMINOPHEN ATTENUATES PULMONARY VASCULAR RESISTANCE AND PULMONARY ARTERIAL PRESSURE AND INHIBITS CARDIOVASCULAR COLLAPSE IN A PORCINE MODEL OF ENDOTOXEMIA.

Resistance training reduced luteinising hormone levels in postmenopausal women in a substudy of a randomised controlled clinical trial: A clue to how resistance training reduced vasomotor symptoms.

Fever During Localized Inflammation in Mice Is Elicited by a Humoral Pathway and Depends on Brain Endothelial Interleukin-1 and Interleukin-6 Signaling and Central EP3 Receptors

Microglial activation elicits a negative affective state through prostaglandin-mediated modulation of striatal neurons

Hippocampal Function Is Impaired by a Short-Term High-Fat Diet in Mice: Increased Blood-Brain Barrier Permeability and Neuroinflammation as Triggering Events.

Calcitonin gene related peptide α is dispensable for many danger-related motivational responses.

NF-κB signaling in tanycytes mediates inflammation-induced anorexia.

Immune-mediated febrile response in female rats: Role of central hypothalamic mediators

IL-6R expressed on CNS vascular endothelial cells contributes to the development of experimental autoimmune encephalomyelitis in mice.

Collaborators (5)

Anders Blomqvist

Sigrid Nilsson

Anna Mathia Klawonn

Michael Fritz

Emilia Berin

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