David Loane

Assistant Professor of Neuroscience

Trinity College Dublin

Ireland

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David Loane is an Assistant Professor of Neuroscience at Trinity College Dublin, Ireland. His research focuses on traumatic brain injury, neuroinflammation, and the brain-gut axis, as evidenced by his recent publications addressing the dynamics of astrocytes and the immune response following injury. He explores targeted therapeutic approaches to improve outcomes and conducts extensive assessments of sensorimotor functions in various experimental models.

Articles (11)

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2023

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2021

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2021

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2021

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2021

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2021

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2020

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2020

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2020

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2020

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2020

Collaborators (6)

J. Marc Simard

University of Maryland School of Medicine

UNITED STATES

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Ilia V. Baskakov

University of Maryland School of Medicine

UNITED STATES

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Linda Noble-Haeusslein

Professor

University of Texas at Austin

UNITED STATES

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Rebecca Henry

Royal Society - SFI University Research Fellow/Assistant Professor

University College Dublin

IRELAND

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Alan I. Faden

Associate Dean

University of Maryland

UNITED STATES

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Candace Floyd

Professor

Emory University

UNITED STATES

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David Loane

Research Interests

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Articles (11)

Region-Specific Homeostatic Identity of Astrocytes Is Essential for Defining Their Response to Pathological Insults

Traumatic Brain Injury Induces cGAS Activation and Type I Interferon Signaling in Aged Mice.

Brain-gut axis dysfunction in the pathogenesis of traumatic brain injury.

Targeting chronic and evolving neuroinflammation following traumatic brain injury to improve long-term outcomes: insights from microglial-depletion models.

Pre-Clinical Common Data Elements for Traumatic Brain Injury Research: Progress and Use Cases.

Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits.

Delayed microglial depletion after spinal cord injury reduces chronic inflammation and neurodegeneration in the brain and improves neurological recovery in male mice.

Longitudinal Assessment of Sensorimotor Function after Controlled Cortical Impact in Mice: Comparison of Beamwalk, Rotarod, and Automated Gait Analysis Tests.

Putative mGluR4 positive allosteric modulators activate Gi-independent anti-inflammatory mechanisms in microglia.

Early or Late Bacterial Lung Infection Increases Mortality After Traumatic Brain Injury in Male Mice and Chronically Impairs Monocyte Innate Immune Function.

Enhanced Akt/GSK-3β/CREB signaling mediates the anti-inflammatory actions of mGluR5 positive allosteric modulators in microglia and following traumatic brain injury in male mice

Collaborators (6)

J. Marc Simard

Ilia V. Baskakov

Linda Noble-Haeusslein

Rebecca Henry

Alan I. Faden

Candace Floyd

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