David Macias

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Profesor Ayudante Doctor (Assistant Professor)

Universidad de Sevilla

Spain

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About

David Macias is an Assistant Professor at Universidad de Sevilla, Spain. His research areas include cancer biology, with a focus on tumorigenesis, cellular senescence, and the role of microglia in glial dysfunction. Recent articles authored by him explore the mechanisms of lung cancer progression and potential therapeutic targets.

Recent Grants

Grant: Close

Hypoxic regulation of the senescent secretome in age-related pathologies

Open Date: 2023-09-01

Close Date: 2025-08-01

Grant: Close

Metabolic induction of microglial quiescence

Open Date: 2022-12-01

Close Date: 2025-12-01

Grant: Close

Hypoxic regulation of the senescent secretome in age-related pathologies

Open Date: 2022-09-01

Close Date: 2025-08-01

Grant: Close

Hypoxic regulation of senescent cells secretome in lung cancer and Alzheimer's disease

Open Date: 2021-01-01

Close Date: 2023-01-01

Grant: Close

Desarrollo de vectores lentivirales para terapia génica del VIH-1

Open Date: 2005-01-01

Close Date: 2009-01-01

Articles (13)

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2024

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2024

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2024

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2024

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2023

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2023

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2023

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2022

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2021

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2021

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2021

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2020

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2020

Collaborators (12)

Juan José Pérez-Moreno

Associate Professor

Universidad de Sevilla

SPAIN

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Randall Johnson

Karolinska Institutet

SWEDEN

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Antonia Gutiérrez Pérez

Full Professor of Cell Biology

University of Malaga

SPAIN

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Laura Trujillo-Estrada

Profesor sustituto interino

University of Malaga

SPAIN

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Manuel Collado

Head of the Cell Senescence, Cancer and Aging Laboratory

Centro Nacional de Biotecnología

SPAIN

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Mark J. W. McPhail

UNITED KINGDOM

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Jose L Nieto-Gonzalez

Associate Professor

Universidad de Málaga Facultad de Medicina

SPAIN

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Alexander Wood

University of Western Australia

AUSTRALIA

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Alberto Pascual

SPAIN

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James Joseph

Academic, Lecturer (Teaching and Research)

University of Dundee

UNITED KINGDOM

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Samir Morsli

Karolinska Institutet

SWEDEN

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Daniel J Simpson

Research Temporary Professional

Mayo Clinic

UNITED STATES

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David Macias

Research Interests

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About

Recent Grants

Articles (13)

Microglia mitochondrial complex I deficiency during development induces glial dysfunction and early lethality

An Indocyanine Green‐Based Nanoprobe for <i>In Vivo</i> Detection of Cellular Senescence

Microglia mitochondrial complex I deficiency during development induces glial dysfunction and early lethality

Inhibition of lung tumorigenesis by transient reprogramming in cancer cells

Clearance of senescent macrophages ameliorates tumorigenesis in KRAS-driven lung cancer

A tumor-promoting senescent secretome triggered by platinum chemotherapy exploits a targetable TGFβR1/Akt-mTOR axis in lung cancer

Failed reprogramming of transformed cells due to induction of apoptosis and senescence impairs tumor progression in lung cancer

A tumour-promoting senescent secretome triggered by platinum chemotherapy exploits a targetable TGFβR1/Akt-mTOR axis in lung cancer

Endothelial cell regulation of systemic haemodynamics and metabolism acts through the HIF transcription factors

Targeting HIF2α-ARNT hetero-dimerisation as a novel therapeutic strategy for pulmonary arterial hypertension

Endothelial cell regulation of systemic haemodynamics and metabolism acts through the HIF transcription factors

2-Hydroxyglutarate Metabolism Is Altered in an in vivo Model of LPS Induced Endotoxemia

Galacto-conjugation of Navitoclax as an efficient strategy to increase senolytic specificity and reduce platelet toxicity.

Collaborators (12)

Juan José Pérez-Moreno

Randall Johnson

Antonia Gutiérrez Pérez

Laura Trujillo-Estrada

Manuel Collado

Mark J. W. McPhail

Jose L Nieto-Gonzalez

Alexander Wood

Alberto Pascual

James Joseph

Samir Morsli

Daniel J Simpson

Make your next move.

David Macias

Research Interests

Explore related searches

Contact this professor

About

Recent Grants

Articles (13)

Microglia mitochondrial complex I deficiency during development induces glial dysfunction and early lethality

An Indocyanine Green‐Based Nanoprobe for <i>In Vivo</i> Detection of Cellular Senescence

Microglia mitochondrial complex I deficiency during development induces glial dysfunction and early lethality

Inhibition of lung tumorigenesis by transient reprogramming in cancer cells

Clearance of senescent macrophages ameliorates tumorigenesis in KRAS-driven lung cancer

A tumor-promoting senescent secretome triggered by platinum chemotherapy exploits a targetable TGFβR1/Akt-mTOR axis in lung cancer

Failed reprogramming of transformed cells due to induction of apoptosis and senescence impairs tumor progression in lung cancer

A tumour-promoting senescent secretome triggered by platinum chemotherapy exploits a targetable TGF&beta;R1/Akt-mTOR axis in lung cancer

Endothelial cell regulation of systemic haemodynamics and metabolism acts through the HIF transcription factors

Targeting HIF2α-ARNT hetero-dimerisation as a novel therapeutic strategy for pulmonary arterial hypertension

Endothelial cell regulation of systemic haemodynamics and metabolism acts through the HIF transcription factors

2-Hydroxyglutarate Metabolism Is Altered in an in vivo Model of LPS Induced Endotoxemia

Galacto-conjugation of Navitoclax as an efficient strategy to increase senolytic specificity and reduce platelet toxicity.

Collaborators (12)

Juan José Pérez-Moreno

Randall Johnson

Antonia Gutiérrez Pérez

Laura Trujillo-Estrada

Manuel Collado

Mark J. W. McPhail

Jose L Nieto-Gonzalez

Alexander Wood

Alberto Pascual

James Joseph

Samir Morsli

Daniel J Simpson

Make your next move.

A tumour-promoting senescent secretome triggered by platinum chemotherapy exploits a targetable TGFβR1/Akt-mTOR axis in lung cancer