David N. Cooper

Professor of Human Molecular Genetics

Cardiff University School of Medicine

United Kingdom

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David N. Cooper is a Professor of Human Molecular Genetics at Cardiff University School of Medicine, located in the United Kingdom. His research focuses on human genetics, with recent articles exploring topics such as predicting drug side effects through genetic priority scores, the genetic basis of chronic diseases, and the evolutionary adaptations of primate species. Cooper's work significantly contributes to understanding the genetic underpinnings of complex diseases and their associated risks.

Articles (12)

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2025

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2024

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2023

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2023

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2023

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2023

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2022

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2022

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2021

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2021

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2021

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2021

Collaborators (6)

Jeffrey M. Friedman

Rockefeller University

UNITED STATES

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Marie Verbanck

Assistant Professor

Université Paris-Saclay

FRANCE

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Matthew Mort

Cardiff University

UNITED KINGDOM

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Ben Omega Petrazzini

University of Oxford

UNITED KINGDOM

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Claude Ferec

Professor of Genetic, MD

Université de Bretagne Occidentale

FRANCE

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Maria J. Prata

University of Porto

PORTUGAL

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David N. Cooper

Research Interests

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About

Articles (12)

Development of a genetic priority score to predict drug side effects using human genetic evidence

Expanding drug targets for 112 chronic diseases using a machine learning-assisted genetic priority score

Inferring the genetic relationship between brain imaging-derived phenotypes and risk of complex diseases by Mendelian randomization and genome-wide colocalization

Lineage-specific accelerated sequences underlying primate evolution

Identification of protease-sensitive but not misfolding PNLIP variants in familial and hereditary pancreatitis

The PRSS3P2 and TRY7 deletion copy number variant modifies risk for chronic pancreatitis

Functional genomics analysis reveals the evolutionary adaptation and demographic history of pygmy lorises

Trypsinogen (PRSS1 and PRSS2) gene dosage correlates with pancreatitis risk across genetic and transgenic studies: a systematic review and re-analysis

Chronic Pancreatitis: The True Pathogenic Culprit within the SPINK1 N34S-Containing Haplotype Is No Longer at Large

Atypical NF1 Microdeletions: Challenges and Opportunities for Genotype/Phenotype Correlations in Patients with Large NF1 Deletions

The genetic structure of the Turkish population reveals high levels of variation and admixture

Common polymorphic OTC variants can act as genetic modifiers of enzymatic activity

Collaborators (6)

Jeffrey M. Friedman

Marie Verbanck

Matthew Mort

Ben Omega Petrazzini

Claude Ferec

Maria J. Prata

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