David Orlicky

Associate Professor

United States

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About

David Orlicky is an Associate Professor at the University of Colorado Anschutz Medical Center in the United States. His research areas include the mechanical and biochemical properties of connective tissues, inflammatory responses following burn injuries, and the molecular mechanisms underlying various disease states in murine models. Recent publications highlight his work on the tensile behavior of the murine ulterosacral ligament, post-burn lung inflammation, and metabolic dysregulation associated with liver disease.

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2025

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2022

Collaborators (16)

Joshua Johnson

Assistant Professor

University of Colorado Boulder

UNITED STATES

David Orlicky

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Articles (20)

Ag-driven CD8+ T cell clonal expansion is a prominent feature of MASH in humans and mice

Effect of pregnancy, vaginal delivery, postpartum remodeling, and age on the tensile behavior and biochemical composition of the murine ulterosacral ligament (USL)

Post-burn Lung Inflammation Is Associated With Induction of Pulmonary Cathelicidin-related Antimicrobial Peptide and S100a8 in Mice

Gene-dose effect of the glutathione biosynthesis gene on ascorbate deficiency in mice

Cell Models of Castration Resistant and High Dose Testosterone-Resistant Prostate Cancer Recapitulate the Heterogeneity of Response Observed in Clinical Practice

Activation of AMPD2 drives metabolic dysregulation and liver disease in mice with hereditary fructose intolerance

Inflammatory Cells in Control and Prolapsed Uterosacral Ligament Tissue

Overexpression of TNFα in TNF∆ARE+/− mice increases hepatic periportal inflammation and alters bile acid signaling in mice

Quantifying Protein Acetylation in Diabetic Nephropathy from Formalin‐Fixed Paraffin‐Embedded Tissue

Reducing the excessive inflammation after burn injury in aged mice by maintaining a healthier intestinal microbiome

IL-6 mediates the hepatic acute phase response after prerenal azotemia in a clinically defined murine model

Expression of circadian regulatory genes is dysregulated by increased cytokine production in mice subjected to concomitant intestinal injury and parenteral nutrition

Publisher Correction: Pharmacologic inhibition of HNF4α prevents parenteral nutrition associated cholestasis in mice

Estrogen and Androgen Receptor Status in Uterosacral Ligaments of Women with Pelvic Organ Prolapse Stratified by the Pelvic Organ Prolapse Histology Quantification System

Pharmacologic inhibition of HNF4α prevents parenteral nutrition associated cholestasis in mice

Innate Immune Zonation in the Liver: NF-κB (p50) Activation and C-Reactive Protein Expression in Response to Endotoxemia Are Zone Specific

High Fructose Corn Syrup Accelerates Kidney Disease and Mortality in Obese Mice with Metabolic Syndrome

Adrenal gene expression dynamics support hibernation in 13-lined ground squirrels

The autophagic protein p62 is a target of reactive aldehydes in human and murine cholestatic liver disease

The role of FXR and TGR5 in reversing and preventing progression of Western diet–induced hepatic steatosis, inflammation, and fibrosis in mice

Collaborators (16)

Joshua Johnson

Laura Graham

Dean Tolan

Sarah Faubel

Clyde J. Wright

Richard Johnson

Sarah Calve

Sandra L. Martin

Luciano Adorini

Callan Luetkemeyer

Beth Tamburini

Virginia Ferguson

Masanari Kuwabara

Ronald Sokol

Matthew Burchill

Ana Andres-Hernando

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