David Rasicci

Assistant Professor

West Virginia University

United States

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David Rasicci is an Assistant Professor at West Virginia University, USA. His research areas include cardiac myopathy, anatomical variations, and educational methodologies in human anatomy. Recent publications highlight the biochemical mechanisms underlying alcoholic cardiomyopathy, the role of anatomical education in interprofessional settings, and the implications of genetic mutations in cardiac muscle function.

Articles (10)

Year:

2025

Year:

2025

Year:

2025

Year:

2025

Year:

2024

Year:

2024

Year:

2024

Ischemic heart failure is associated with impairments in contractile performance of the heart. This study revealed that cardiac myosin isolated from patients with ischemic heart failure had reduced mechanical activity, which correlated with the impaired clinical phenotype of the patients. The results suggest that restoring myosin function with pharmacological intervention may be a viable method for therapeutic intervention.

Year:

2023

Year:

2023

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Sivaraj Sivaramakrishnan

UNITED STATES

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Wen Ma

Assistant Professor

University of Vermont

UNITED STATES

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David Rasicci

Research Interests

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Articles (10)

Early-Stage Alcoholic Cardiomyopathy Highlighted by Metabolic Remodeling, Oxidative Stress, and Cardiac Myosin Dysfunction in Male Rats

Hairy Esophagus: Incidental Finding of Esophageal Reconstruction Surgery in a Gross Anatomy Laboratory Stimulates Near-Peer Learning and Interprofessional Education

A constellation of anatomical variation: middle scalene, wrist extensor, and aortic arch variants share embryological origins in cervical somites

Retrospective Analysis of a Mature Cystic Teratoma Discovered in the Anatomy Laboratory: Cross-Curricular Integration of Gross Anatomy, Histology, and Embryology

Dilated cardiomyopathy mutation in beta-cardiac myosin enhances actin activation of the power stroke and phosphate release

Tail length and E525K dilated cardiomyopathy mutant alter human β-cardiac myosin super-relaxed state

Geographical Impact of Human Gift Registries in West Virginia: A Model for Centralized Resources in Human Anatomy Education

Cardiac myosin motor deficits are associated with left ventricular dysfunction in human ischemic heart failure

Myosin loop-4 is critical for optimal tropomyosin repositioning on actin during muscle activation and relaxation

Dilated cardiomyopathy mutation (E525K) in human beta-cardiac myosin enhances actin-activated phosphate release but stabilizes the auto-inhibited super-relaxed state

Collaborators (10)

Kenneth S. Campbell

Jinghua Ge

David M. Warshaw

J. Andrew McCammon

C. M. Yengo

Michael Previs

Michael J. Rynkiewicz

Yuval Silberman

Sivaraj Sivaramakrishnan

Wen Ma

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