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David Wu

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Life Science Research Professional

Stanford University

United States

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About

David Wu is a Life Science Research Professional at Stanford University in the United States. His research areas focus on cardiomyocyte function and disease mechanisms, particularly in relation to long COVID, arrhythmogenesis, and induced pluripotent stem cell technologies. Recent publications include investigations into cardiac dysfunction linked to CCL2 in long COVID and the generation of iPSC lines from patients with various cardiovascular conditions.

Recent Grants

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Utilizing an iPSC-Based Ischemia Model to Elucidate Racial Disparities in Cardiovascular Disease

Open Date: 2023-08-01

Close Date: 2024-08-01

Articles (14)

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Collaborators (8)

Nazish Sayed

Stanford University

UNITED STATES

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José Jalife

Senior Investigator/Director

University of Michigan

UNITED STATES

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Baohui Xu

Stanford University

UNITED STATES

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Ioannis Karakikes

Associate Professor

Stanford University

UNITED STATES

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Melinda L Telli

Professor of Medicine

Stanford University School of Medicine

UNITED STATES

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Paul Cheng

Stanford University

UNITED STATES

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Philip S Tsao

Stanford University

UNITED STATES

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Dilip Thomas

Assistant Professor

University of Galway

IRELAND

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David Wu

Research Interests

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About

Recent Grants

Articles (14)

Generation of two homozygous iPSC lines carrying variants of uncertain significance in LMNA associated with cardiomyopathy

RUNX1-driven endothelial-to-mesenchymal transition contributes to remodelling in LMNA cardiomyopathy

Generation of an induced pluripotent stem cell line from a patient with Loeys–Dietz syndrome

Harnessing iPSCs, 3D organoids, and multiomics to model rare vascular diseases: Emerging new approach methodologies

Investigating the Risk of Arrhythmogenesis Associated With Fentanyl Abuse Using Human and Mouse Cardiomyocytes

Generation of induced pluripotent stem cell line from a patient with long COVID

Exploration of Underlying Mechanisms of Vascular Ehlers-Danlos Syndrome in Patients' iPSCs-Derived Endothelial Cells

Multiscale profiling of tyrosine kinase inhibitor cardiotoxicity reveals mechanosensitive ion channel PIEZO1 as cardioprotective

Generation of an induced pluripotent stem cell line from a patient with Varicose veins

Immunosuppression Drugs Exhibit Differential Effects on Endothelial Cell Function

CCL2-mediated endothelial injury drives cardiac dysfunction in long COVID

Generation of two iPSC lines from vascular Ehlers-Danlos Syndrome (vEDS) patients carrying a missense mutation in COL3A1 gene

Generation of two iPSC lines from dilated cardiomyopathy patients with pathogenic variants in the SCN5A gene

Generation of induced pluripotent stem cell line from a patient suffering from arterial calcification due to deficiency of CD73 (ACDC)

Collaborators (8)

Nazish Sayed

José Jalife

Baohui Xu

Ioannis Karakikes

Melinda L Telli

Paul Cheng

Philip S Tsao

Dilip Thomas

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