Dietmar J. Manstein

Professor & Direktor

Medizinische Hochschule Hannover

Germany

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Articles (13)

Year:

2024

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2024

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2024

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2022

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (4)

Daniel Mulvihill

Professor of Cell and Molecular Biology

UNITED KINGDOM

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VINCENZO LOMBARDI

Professor

University of Florence

ITALY

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Amrita Rai

Max Planck Institute of Molecular Physiology

GERMANY

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Marco Caremani

Professore Associato

University of Florence

ITALY

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Dietmar J. Manstein

Research Interests

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Articles (13)

Biochemical characterization of cardiac α‐actin mutations A21V and D26N implicated in hypertrophic cardiomyopathy

The non-muscle actinopathy-associated mutation E334Q in cytoskeletal γ-actin perturbs interaction of actin filaments with myosin and ADF/cofilin family proteins

The non-muscle actinopathy-associated mutation E334Q in cytoskeletal γ-actin perturbs interaction of actin filaments with myosin and ADF/cofilin family proteins

Distinct actin–tropomyosin cofilament populations drive the functional diversification of cytoskeletal myosin motor complexes

Structural and Biochemical Characterization of a Dye-Decolorizing Peroxidase from Dictyostelium discoideum

Assessment of the Contribution of a Thermodynamic and Mechanical Destabilization of Myosin-Binding Protein C Domain C2 to the Pathomechanism of Hypertrophic Cardiomyopathy-Causing Double Mutation MYBPC3Δ25bp/D389V

CORE-MD II: A fast, adaptive, and accurate enhanced sampling method

Allosteric modulation of cardiac myosin mechanics and kinetics by the conjugated omega‐7,9 trans‐fat rumenic acid

Improvement of image resolution by combining enhanced confocal microscopy and quantum dot triexciton imaging

Muscle myosin performance measured with a synthetic nanomachine reveals a class‐specific Ca2+‐sensitivity of the frog myosin II isoform

Mechanochemical properties of human myosin-1C are modulated by isoform-specific differences in the N-terminal extension

Myosin-18B Regulates Higher-Order Organization of the Cardiac Sarcomere through Thin Filament Cross-Linking and Thick Filament Dynamics

Undefeated-Changing the phenamacril scaffold is not enough to beat resistant Fusarium.

Collaborators (4)

Daniel Mulvihill

VINCENZO LOMBARDI

Amrita Rai

Marco Caremani

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