DOLORES PRIETO

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Universidad Complutense de Madrid

Spain

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Professor Dolores Prieto is a faculty member at Universidad Complutense de Madrid, Spain. Her research focuses on the epidemiology of urolithiasis, obesity-related healthcare burdens, and endothelial dysfunction in relation to cardiovascular health. Recent publications include studies on the hospitalization burden of kidney stones and the impact of metabolic comorbidities, emphasizing the complex interplay between obesity, urolithiasis, and vascular function.

Recent Grants

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Renal endothelial dysfunction, oxidative stress and obesity-associated endothelial dysfunction

Open Date: 2017-01-01

Close Date: 2019-12-01

Grant: Close

"Obesity and Kidney disease: role of mitochondria and ER stress in obesity-associated vascular dysfunction"

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Articles (13)

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2025

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2024

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2024

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2023

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2023

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2022

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2022

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (7)

Alejandro Gutierrez

University of Nevada Reno

UNITED STATES

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Alfonso Muriel

Universidad de Alcalá

SPAIN

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ANGEL GIL DE MIGUEL

Universidad Rey Juan Carlos

SPAIN

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Estéfano Pinilla

Aarhus University

DENMARK

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Mercedes Muñoz Picos

Profesora Asociada

Universidad Complutense de Madrid

SPAIN

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Martín Santos

Universidad Francisco de Vitoria

SPAIN

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Cristina Contreras

Assistant Professor

Universidad Complutense de Madrid

SPAIN

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DOLORES PRIETO

Research Interests

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About

Recent Grants

Articles (13)

Impact of Obesity on Hospitalization Burden of Urolithiasis in Spain Between 1997 and 2021

Cardiovascular and Cerebrovascular Morbidity in Patients with Urolithiasis: An Epidemiological Approach Based on Hospitalization Burden Data from 1997 to 2021

Activation of mitoKATP channels induces penile vasodilation and inhibits mitochondrial respiration and ROS production: Role of NO

Hospitalization Burden of Patients with Kidney Stones and Metabolic Comorbidities in Spain during the Period 2017–2020

Calcium handling coupled to the endothelin ETA and ETB receptor-mediated vasoconstriction in resistance arteries: Differential regulation by PI3K, PKC and RhoK

Hyperoxaluria Induces Endothelial Dysfunction in Preglomerular Arteries, Involvement of Oxidative Stress

Endothelial Dysfunction: An Intermediate Clinical Feature between Urolithiasis and Cardiovascular Diseases

Activation of AMP kinase ameliorates kidney vascular dysfunction, oxidative stress and inflammation in rodent models of obesity

AMPK, metabolism, and vascular function

Urolithiasis Develops Endothelial Dysfunction as a Clinical Feature

Endothelial KCa1.1 and KCa3.1 channels mediate rat intrarenal artery endothelium‐derived hyperpolarization response

Metabolic syndrome inhibits store-operated Ca2+ entry and calcium-induced calcium-release mechanism in coronary artery smooth muscle

Activation of the AMP-related kinase (AMPK) induces renal vasodilatation and downregulates Nox-derived reactive oxygen species (ROS) generation

Collaborators (7)

Alejandro Gutierrez

Alfonso Muriel

ANGEL GIL DE MIGUEL

Estéfano Pinilla

Mercedes Muñoz Picos

Martín Santos

Cristina Contreras

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