Durgesh Tiwari

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Assistant Professor

Cincinnati Children's Hospital Medical Center

United States

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Durgesh Tiwari is an Assistant Professor at Cincinnati Children's Hospital Medical Center in the United States. His research focuses on neuroscience and epilepsy, particularly the role of microRNAs and neural circuits in disease etiology and treatment. Recent articles authored by Dr. Tiwari explore topics such as dendritic spine regulation, the effects of sex hormones on seizure susceptibility, and the implications of mTOR pathways in epileptogenesis.

Recent Grants

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Postdoctoral fellowship from American Epilepsy Society

Open Date: 2017-07-01

Close Date: 2018-07-01

Articles (10)

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2025

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2023

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2023

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2023

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2020

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2020

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2020

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2020

Collaborators (3)

Michael Williams

Associate Professor

Cincinnati Children's Hospital Medical Center

UNITED STATES

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Nicole Oatman

Ghent University

BELGIUM

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Stefanie Schirmeier

Professor for Zoology and Animal Physiology

TU Dresden

GERMANY

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Durgesh Tiwari

Research Interests

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Recent Grants

Articles (10)

How Soon is Soon Enough? Consideration of Timing and Disease Etiology for microRNAs in Epilepsy

MiR-324-5p inhibition after intrahippocampal kainic acid-induced status epilepticus does not prevent epileptogenesis in mice

Age-Dependent Regulation of Dendritic Spine Density and Protein Expression in Mir324 KO Mice

Estradiol- and Progesterone-Associated Changes in microRNA-Induced Silencing and Reduced Antiseizure Efficacy of an Antagomir in Female Mice

GABA<inf>A</inf> Alpha 2,3 Modulation Improves Select Phenotypes in a Mouse Model of Fragile X Syndrome

AMPK-Regulated Astrocytic Lactate Shuttle Plays a Non-Cell-Autonomous Role in Neuronal Survival

mTOR-driven neural circuit changes initiate an epileptogenic cascade.

Violet-light suppression of thermogenesis by opsin 5 hypothalamic neurons.

The potassium channel Kv4.2 regulates dendritic spine morphology, electroencephalographic characteristics and seizure susceptibility in mice.

PI3K isoform-selective inhibition in neuron-specific PTEN-deficient mice rescues molecular defects and reduces epilepsy-associated phenotypes.

Collaborators (3)

Michael Williams

Nicole Oatman

Stefanie Schirmeier

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