Dwight Bergles

Professor

Johns Hopkins University School of Medicine

United States

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Dwight Bergles is a Professor at the Johns Hopkins School of Medicine in the United States. His research focuses on neurodevelopmental disorders, synaptic plasticity, and the role of oligodendrocytes in brain health through recent studies involving neuronal models and calcium dynamics. He has contributed to understanding neuronal development and connectivity, as well as the metabolic coupling between oligodendrocytes and axons.

Articles (16)

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2025

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GERMANY

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Austin Graves

Research Assistant Professor

Johns Hopkins University

UNITED STATES

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Aiman S. Saab

SNSF Eccellenza Professor

University of Zurich

SWITZERLAND

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Jonathan Ling

Assistant Professor

Johns Hopkins University School of Medicine

UNITED STATES

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Calvin Kersbergen

National Institutes of Health

UNITED STATES

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Luca Ravotto

University of Zurich

SWITZERLAND

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Emily P Harrington

Assistant Professor

The Ohio State University College of Medicine

UNITED STATES

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Torben Ruhwedel

Max Planck Institute for Multidisciplinary Sciences

GERMANY

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atsushi Kamiya

Professor

Johns Hopkins University School of Medicine

UNITED STATES

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Hauke Werner

GERMANY

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Dwight Bergles

Research Interests

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About

Articles (16)

Altered development and network connectivity in a human neuronal model of 15q11.2 deletion-related neurodevelopmental disorders

Transcriptional profiles of mouse oligodendrocyte precursor cells across the lifespan

Oligodendrocyte–axon metabolic coupling is mediated by extracellular K+ and maintains axonal health

Correction: Transient Receptor Potential (TRP) Channels in Cochlear Function: Looking Beyond Mechanotransduction

Transient Receptor Potential (TRP) Channels in Cochlear Function: Looking Beyond Mechanotransduction

Multicore fiber optic imaging reveals that astrocyte calcium activity in the mouse cerebral cortex is modulated by internal motivational state

Cross-modality supervised image restoration enables nanoscale tracking of synaptic plasticity in living mice

Preservation of developmental spontaneous activity enables early auditory system maturation in deaf mice.

Loss of Astrocytic µ Opioid Receptors Exacerbates Aversion Associated with Morphine Withdrawal in Mice: Role of Mitochondrial Respiration

MHC class I and MHC class II reporter mice enable analysis of immune oligodendroglia in mouse models of multiple sclerosis

Norepinephrine modulates calcium dynamics in cortical oligodendrocyte precursor cells promoting proliferation during arousal in mice

Developmental spontaneous activity promotes formation of sensory domains, frequency tuning and proper gain in central auditory circuits.

Olig2Ablation in Immature Oligodendrocytes Does Not Enhance CNS Myelination and Remyelination

Author Correction: Deep-learning two-photon fiberscopy for video-rate brain imaging in freely-behaving mice

Deep-learning two-photon fiberscopy for video-rate brain imaging in freely-behaving mice

Stage-specific control of oligodendrocyte survival and morphogenesis by TDP-43

Collaborators (14)

Christa Habela

Richard Huganir

yun guan

Jin Kang

Amit Agarwal

Austin Graves

Aiman S. Saab

Jonathan Ling

Calvin Kersbergen

Luca Ravotto

Emily P Harrington

Torben Ruhwedel

atsushi Kamiya

Hauke Werner

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