Edgar Walters

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The University of Texas Health Science Center at Houston John P and Katherine G McGovern Medical School

United States

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About

Professor Edgar Walters is a faculty member at The University of Texas Health Science Center at Houston John P and Katherine G McGovern Medical School in the United States. His research primarily focuses on nociceptor hyperexcitability, pain mechanisms, and the cellular signaling pathways involved in pain perception. Recent publications include studies on the electrophysiological alterations in nociceptors and their contributions to pain-related behaviors following injury.

Recent Grants

Grant: Close

Mechanisms in primary nociceptors that drive ongoing activity and ongoing pain

Open Date: 2019-04-15

Close Date: 2024-03-31

Grant: Close

MECHANISMS OF OPIOID RESISTANCE AFTER SPINAL CORD INJURY

Open Date: 2018-07-31

Close Date: 2022-07-30

Grant: Close

Mechanisms of cAMP signaling that drive spontaneous activity in nociceptors

Open Date: 2015-08-15

Close Date: 2025-07-31

Grant: Close

Contributions of inflammatory mediators in chronic SCI

Open Date: 2013-07-01

Close Date: 2015-06-30

Grant: Close

Novel Target for Ameliorating Pain and Other Problems after SCI: Spontaneous Activity in Nociceptors

Open Date: 2012-09-15

Close Date: 2016-03-14

Articles (12)

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2025

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2023

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2022

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2022

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2022

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2021

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2020

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UNITED STATES

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Alexis Guillaume Bavencoffe

Assistant Professor

The University of Texas Health Science Center at Houston

UNITED STATES

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Edgar Walters

Research Interests

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About

Recent Grants

Articles (12)

Widespread hyperexcitability of nociceptor somata outlasts enhanced avoidance behavior after incision injury

Induction of long-term hyperexcitability by memory-related cAMP signaling in isolated nociceptor cell bodies

Persistent nociceptor hyperactivity as a painful evolutionary adaptation

Readiness of nociceptor cell bodies to generate spontaneous activity results from background activity of diverse ion channels and high input resistance

Macrophage Migration Inhibitory Factor (MIF) Makes Complex Contributions to Pain-Related Hyperactivity of Nociceptors after Spinal Cord Injury

Electrophysiological Alterations Driving Pain-Associated Spontaneous Activity in Human Sensory Neuron Somata Parallel Alterations Described in Spontaneously Active Rodent Nociceptors

Major Differences in Transcriptional Alterations in Dorsal Root Ganglia between Spinal Cord Injury and Peripheral Neuropathic Pain Models

Strong inferences about pain in invertebrates require stronger evidence

Serotonin enhances depolarizing spontaneous fluctuations, excitability, and ongoing activity in isolated rat DRG neurons via 5-HT4 receptors and cAMP-dependent mechanisms

Oral Dimethyl Fumarate Reduces Peripheral Neuropathic Pain in Rodents via NFE2L2 Antioxidant Signaling.

Depolarization-Dependent C-Raf Signaling Promotes Hyperexcitability and Reduces Opioid Sensitivity of Isolated Nociceptors after Spinal Cord Injury

Interleukin-10 resolves pain hypersensitivity induced by cisplatin by reversing sensory neuron hyperexcitability

Collaborators (6)

Peter Grace

Patrick Dougherty

Carmen Dessauer

Ryan Michael Cassidy

Robert North

Alexis Guillaume Bavencoffe

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