Erik Johnson

Assistant Professor

Emory University School of Medicine

United States

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Articles (9)

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2024

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2024

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2023

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2023

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2023

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2022

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2020

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2020

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2020

Collaborators (11)

Alisha Daniels

MD, MHA; Executive Research Director/Assistant Professor

Washington University in St. Louis

UNITED STATES

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Aliza Wingo

Associate Professor

Emory University

UNITED STATES

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Blaine Roberts

Associate Professor

Emory University

UNITED STATES

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Nick Fox

Professor and Head of Department

UCL Institute of Neurology, University College London

UNITED KINGDOM

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Lenora Higginbotham

Emory University

UNITED STATES

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Chris Gaiteri

Assistant professor

Rush University Medical Center

UNITED STATES

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Andrew Saykin

Professor and Director, Center for Neuroimaging and Indiana Alzheimer's Disease Research Center

Indiana University School of Medicine

UNITED STATES

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Jeanne Paz

University of California

UNITED STATES

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Charlene Supnet

Associate Professor

Washington University in Saint Louis School of Medicine

UNITED STATES

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Brian Gordon

Assistant Professor

Washington University in Saint Louis School of Medicine

UNITED STATES

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Allan I. Levey

Emory University School of Medicine

UNITED STATES

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Erik Johnson

Research Interests

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Articles (9)

Author Correction: Serum proteomics reveal APOE-ε4-dependent and APOE-ε4-independent protein signatures in Alzheimer’s disease

Serum proteomics reveal APOE-ε4-dependent and APOE-ε4-independent protein signatures in Alzheimer’s disease

A protein panel in cerebrospinal fluid for diagnostic and predictive assessment of Alzheimer’s disease

Cerebrospinal fluid proteomics define the natural history of autosomal dominant Alzheimer’s disease

Quantitative Mass Spectrometry Analysis of Cerebrospinal Fluid Protein Biomarkers in Alzheimer’s Disease

Large-scale deep multi-layer analysis of Alzheimer’s disease brain reveals strong proteomic disease-related changes not observed at the RNA level

Integrated proteomics reveals brain-based cerebrospinal fluid biomarkers in asymptomatic and symptomatic Alzheimer’s disease

Behavioral and neural network abnormalities in human APP transgenic mice resemble those of App knock-in mice and are modulated by familial Alzheimer's disease mutations but not by inhibition of BACE1.

Large-scale proteomic analysis of Alzheimer’s disease brain and cerebrospinal fluid reveals early changes in energy metabolism associated with microglia and astrocyte activation

Collaborators (11)

Alisha Daniels

Aliza Wingo

Blaine Roberts

Nick Fox

Lenora Higginbotham

Chris Gaiteri

Andrew Saykin

Jeanne Paz

Charlene Supnet

Brian Gordon

Allan I. Levey

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