Eugenia Trushina

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Mayo Clinic Minnesota

United States

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Professor Eugenia Trushina is a faculty member at Mayo Clinic Minnesota, USA. Her research focuses on the intersection of mitochondrial function and neurodegenerative diseases, with recent articles exploring topics such as autophagy, NAD metabolism, and mitochondrial morphology. She contributes to advancing methodologies for studying Alzheimer's disease through innovative in vitro models and therapeutic strategies.

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Role of Histone Deacetylase in AD Mitochondrial Dysfunction

Open Date: 2011-07-01

Close Date: 2014-06-30

Articles (10)

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2024

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2023

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2023

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2023

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2022

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2022

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2022

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2022

Collaborators (8)

Oliver David Kenneth Maddocks

University of Glasgow

UNITED KINGDOM

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Tracy Young-Pearse

Harvard University

UNITED STATES

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Michael Lazarou

MONASH UNIVERSITY

AUSTRALIA

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Daniel Tennant

University of Birmingham

UNITED KINGDOM

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Alberto Sanz

University of Glasgow

UNITED KINGDOM

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Samuel Lara-Reyna

University of Birmingham

UNITED KINGDOM

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Renata Alambert

Research Assistant Professor

Iowa State University

UNITED STATES

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Tetsushi Kataura

Assistant Professor

University of Tsukuba

JAPAN

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Eugenia Trushina

Research Interests

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About

Recent Grants

Articles (10)

Targeting the autophagy-NAD axis protects against cell death in Niemann-Pick type C1 disease models

NAD depletion mediates cytotoxicity in human neurons with autophagy deficiency

A Comprehensive Approach to Sample Preparation for Electron Microscopy and the Assessment of Mitochondrial Morphology in Tissue and Cultured Cells

Modulation of Mitochondrial Function as a Therapeutic Strategy for Neurodegenerative Diseases

Partial Inhibition of Complex I Restores Mitochondrial Morphology and Mitochondria-ER Communication in Hippocampus of APP/PS1 Mice

Predictive metabolic networks reveal sex- and APOE genotype-specific metabolic signatures and drivers for precision medicine in Alzheimer's disease

Autophagy promotes cell survival by maintaining NAD levels

Prominent Mitochondrial Injury as an Early Event in Heme Protein-Induced Acute Kidney Injury

Mitochondrial complex I as a therapeutic target for Alzheimer's disease

Advances in Recapitulating Alzheimer’s Disease Phenotypes Using Human Induced Pluripotent Stem Cell-Based In Vitro Models

Collaborators (8)

Oliver David Kenneth Maddocks

Tracy Young-Pearse

Michael Lazarou

Daniel Tennant

Alberto Sanz

Samuel Lara-Reyna

Renata Alambert

Tetsushi Kataura

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