Eun Hee Ahn

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Assistant Professor (tenure track)

Hallym University College of Medicine

South Korea

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Dr. Eun Hee Ahn is an Assistant Professor (tenure track) at Hallym University College of Medicine in South Korea. Her research focuses on neurodegenerative diseases, with recent publications examining the roles of ApoE and BDNF in Alzheimer's and Parkinson's disease. She investigates the gut-brain axis and how various molecular interactions impact cognitive dysfunction and neuronal survival.

Recent Grants

Grant: Open

Initiate Tauopathies with Phosphorylated Tau via Inflammatory Cytokines and BDNF/NTN4 Deficiency in the Myenteric neurons.

Open Date: 2022-03-01

Close Date: 2027-02-01

Grant: Close

The gut microbiome imbalance triggers Parkinson's disease through the gut-brain axis via the C/EBPβ/AEP molecular mechanism.

Open Date: 2021-06-01

Close Date: 2022-05-01

Articles (16)

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2025

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2024

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2023

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2023

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2023

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2023

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2023

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2023

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2022

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2021

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (1)

Laura Edgington-Mitchell

MONASH UNIVERSITY

AUSTRALIA

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Eun Hee Ahn

Research Interests

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About

Recent Grants

Articles (16)

ApoE3 R136S binds to Tau and blocks its propagation, suppressing neurodegeneration in mice with Alzheimer's disease

Akkermansia muciniphila improve cognitive dysfunction by regulating BDNF and serotonin pathway in gut-liver-brain axis

The couple of netrin-1/α-Synuclein regulates the survival of dopaminergic neurons via α-Synuclein disaggregation

<i>Helicobacter</i><i> hepaticus</i> augmentation triggers Dopaminergic degeneration and motor disorders in mice with Parkinson's disease

C/EBPβ/AEP is age-dependently activated in Parkinson's disease and mediates α-synuclein in the gut and brain

Development of an a-synuclein positron emission tomography tracer for imaging synucleinopathies

GAP-43 closely interacts with BDNF in hippocampal neurons and is associated with Alzheimer's disease progression

The couple of netrin-1/α-Synuclein regulates the survival of dopaminergic neurons via α-Synuclein disaggregation

UNC5C Receptor Proteolytic Cleavage by Active AEP Promotes Dopaminergic Neuronal Degeneration in Parkinson's Disease

C/EBPβ/AEP Signaling Regulates the Oxidative Stress in Malignant Cancers, Stimulating the Metastasis

Gut inflammation triggers C/EBPβ/δ‐secretase‐dependent gut‐to‐brain propagation of Aβ and Tau fibrils in Alzheimer’s disease

Netrin-1 receptor UNC5C cleavage by active δ-secretase enhances neurodegeneration, promoting Alzheimer’s disease pathologies

Netrin‐1 and its receptor DCC modulate survival and death of dopamine neurons and Parkinson’s disease features

ApoE4 inhibition of VMAT2 in the locus coeruleus exacerbates Tau pathology in Alzheimer's disease

Netrin1 deficiency activates MST1 via UNC5B receptor, promoting dopaminergic apoptosis in Parkinson’s disease

Gut dysbiosis contributes to amyloid pathology, associated with C/EBPβ/AEP signaling activation in Alzheimer’s disease mouse model

Collaborators (1)

Laura Edgington-Mitchell

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