Fabrizia Cesca

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Associate Professor

University of Trieste

Italy

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About

Fabrizia Cesca is an Associate Professor at the University of Trieste, Italy. Her research focuses on neurobiology, specifically investigating the roles of Kidins220 in neural development, sensory processing, and neurogenesis. Recent publications include studies on SINO syndrome phenotype refinement, the impact of Kidins220 expression on behavior in mice, and the modulation of astrocyte responses to neurotrophic factors.

Recent Grants

Grant: Close

Behavioral-cognitive disturbances: a common pathway to heterogeneous synaptopaties

Open Date: 2019-01-01

Close Date: 2020-01-01

Grant: Close

New molecularly targeted therapies for metastatic Uveal Melanoma

Open Date: 2018-01-01

Close Date: 2020-01-01

Grant: Close

Modulation of adult visual cortical plasticity via optogenetic control of gene expression

Open Date: 2015-01-01

Close Date: 2017-01-01

Grant: Close

Novel modulators of sodium channels and neuronal excitability

Open Date: 2014-01-01

Close Date: 2015-01-01

Grant: Close

Study of the role of extracellular vesicles in promoting myogenic differentiation

Open Date: 2014-01-01

Close Date: 2016-01-01

Articles (12)

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2024

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2024

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2023

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2023

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2022

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2021

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2021

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2021

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2021

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2021

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2021

Year:

2020

Collaborators (8)

Isabel Fariñas

University of Valencia

SPAIN

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Giampietro Schiavo

Professor

University College London

UNITED KINGDOM

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Luca MARAGLIANO

Assistant Professor (RTDb)

Università Politecnica delle Marche

ITALY

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ANTONIO JESUS JIMENEZ LARA

Catedrático de Universidad /Professor

University of Malaga

SPAIN

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Davide Pozzi

Humanitas University

ITALY

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Teresa Iglesias

Senior Scientist - Research Professor

Consejo Superior de Investigaciones Cientificas

SPAIN

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Elsebet Østergaard

Associate Professor, PhD

University of Copenhagen

DENMARK

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Pierluigi Valente

ITALY

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Fabrizia Cesca

Research Interests

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About

Recent Grants

Articles (12)

Refining the phenotype of SINO syndrome: A comprehensive cohort report of 14 novel cases

Alterations in KIDINS220/ARMS Expression Impact Sensory Processing and Social Behavior in Adult Mice

Kidins220 sets the threshold for survival of neural stem cells and progenitors to sustain adult neurogenesis

Neuron‐restrictive silencer factor/repressor element 1‐silencing transcription factor (NRSF/REST) controls spatial K+ buffering in primary cortical astrocytes

Kidins220/ARMS modulates brain morphology and anxiety-like traits in adult mice

Kidins220 deficiency causes ventriculomegaly via SNX27-retromer-dependent AQP4 degradation

Graphene Nanoplatelets Render Poly(3-Hydroxybutyrate) a Suitable Scaffold to Promote Neuronal Network Development

A developmental stage- and Kidins220-dependent switch in astrocyte responsiveness to brain-derived neurotrophic factor

The enhancement of activity rescues the establishment of <i>Mecp2</i> null neuronal phenotypes

Stability Studies of New Caged <i>bis</i> ‐deoxy‐coelenterazine Derivatives and Their Potential Use as Cellular pH Probes

Neuroinflammation induces synaptic scaling through IL-1β-mediated activation of the transcriptional repressor REST/NRSF

Engineering REST-Specific Synthetic PUF Proteins to Control Neuronal Gene Expression: A Combined Experimental and Computational Study

Collaborators (8)

Isabel Fariñas

Giampietro Schiavo

Luca MARAGLIANO

ANTONIO JESUS JIMENEZ LARA

Davide Pozzi

Teresa Iglesias

Elsebet Østergaard

Pierluigi Valente

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