Francesco Roberto Simonetti

Assistant Professor

Johns Hopkins University

United States

Research Interests

Explore related searches

Professors in United States Professors in Immunology Immunology professors in United States

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Francesco Roberto Simonetti is an Assistant Professor at Johns Hopkins University in the United States. His research focuses on HIV-1 and SIV reservoirs, mechanisms of viral persistence, and the immune responses of CD4+ T cells. Recent publications include studies on HIV-1 viremia, the neutralization resistance of viral reservoirs, and strategies to enhance natural killer cell-mediated elimination of infected cells.

Articles (10)

Year:

2025

Year:

2025

Year:

2025

Year:

2024

Year:

2024

Year:

2024

Year:

2023

Year:

2023

Year:

2023

Year:

2023

Collaborators (1)

Joyce Jones

Johns Hopkins University School of Medicine

UNITED STATES

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Francesco Roberto Simonetti

Research Interests

Explore related searches

Contact this professor

About

Articles (10)

Proviruses in CD4+ T cells reactive to autologous antigens contribute to nonsuppressible HIV-1 viremia

Factors associated with resistance of HIV-1 reservoir viruses to neutralization by autologous IgG antibodies.

SIV proviruses seeded later in infection are harbored in short-lived CD4<sup>+</sup> T cells.

Cognate antigen engagement induces HIV-1 expression in latently infected CD4<sup>+</sup> T cells from people on long-term antiretroviral therapy.

CD4+ T cells with latent HIV-1 have reduced proliferative responses to T cell receptor stimulation

Bispecific antibodies promote natural killer cell-mediated elimination of HIV-1 reservoir cells

Learning from Persistent Viremia: Mechanisms and Implications for Clinical Care and HIV-1 Cure.

Proviral location affects cognate peptide-induced virus production and immune recognition of HIV-1-infected T cell clones.

The latent reservoir of inducible, infectious HIV-1 does not decrease despite decades of antiretroviral therapy.

Clonally expanded HIV-1 proviruses with 5'-leader defects can give rise to nonsuppressible residual viremia.

Collaborators (1)

Joyce Jones

Make your next move.

Francesco Roberto Simonetti

Research Interests

Explore related searches

Contact this professor

About

Articles (10)

Proviruses in CD4+ T cells reactive to autologous antigens contribute to nonsuppressible HIV-1 viremia

Factors associated with resistance of HIV-1 reservoir viruses to neutralization by autologous IgG antibodies.

SIV proviruses seeded later in infection are harbored in short-lived CD4&lt;sup&gt;+&lt;/sup&gt; T cells.

Cognate antigen engagement induces HIV-1 expression in latently infected CD4&lt;sup&gt;+&lt;/sup&gt; T cells from people on long-term antiretroviral therapy.

CD4+ T cells with latent HIV-1 have reduced proliferative responses to T cell receptor stimulation

Bispecific antibodies promote natural killer cell-mediated elimination of HIV-1 reservoir cells

Learning from Persistent Viremia: Mechanisms and Implications for Clinical Care and HIV-1 Cure.

Proviral location affects cognate peptide-induced virus production and immune recognition of HIV-1-infected T cell clones.

The latent reservoir of inducible, infectious HIV-1 does not decrease despite decades of antiretroviral therapy.

Clonally expanded HIV-1 proviruses with 5'-leader defects can give rise to nonsuppressible residual viremia.

Collaborators (1)

Joyce Jones

Make your next move.

SIV proviruses seeded later in infection are harbored in short-lived CD4<sup>+</sup> T cells.

Cognate antigen engagement induces HIV-1 expression in latently infected CD4<sup>+</sup> T cells from people on long-term antiretroviral therapy.