Gary Charles Sieck

Professor

Mayo Clinic Rochester

United States

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Gary Charles Sieck is a Professor at Mayo Clinic Rochester in the United States. His research areas focus on diaphragm muscle function, neuromotor control, and the impact of various physiological and pharmacological conditions on respiratory mechanics. Recent articles authored by Professor Sieck explore subjects such as the role of diaphragm muscles as pumps, the effects of aging on neuromuscular junctions, and the application of machine learning in identifying eupneic breathing.

Articles (27)

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2025

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2022

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2022

Collaborators (5)

Jin Sun Cho

Associate Professor

Yonsei University College of Medicine

SOUTH KOREA

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Carlos Mantilla

Professor and Chair

Mayo Clinic

UNITED STATES

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Haitao Wang

Assistant Professor

Mayo Clinic Minnesota

UNITED STATES

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Kai He

Assistant Professor

Mayo Clinic

UNITED STATES

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John Halliwill

Assistant, Associate, and Full Professor

University of Oregon

UNITED STATES

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Gary Charles Sieck

Research Interests

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About

Articles (27)

Diaphragm muscle: a pump that can not fail

The value of deep inspirations

Impact of upper cervical spinal cord hemisection on diaphragm neuromotor control

Histamine is a molecular transducer of adaptation to endurance exercise training in humans

TNFα-mediated subcellular heterogeneity of succinate dehydrogenase activity in human airway smooth muscle cells

Chloroquine Causes Aging-like Changes in Diaphragm Neuromuscular Junction Morphology in Mice

Inhibition of TrkB kinase activity impairs autophagy in cervical motor neurons of young but not old mice

Identification of eupneic breathing using machine learning

β-adrenergic stimulation after rewarming does not mitigate hypothermia-induced contractile dysfunction in rat cardiomyocytes

Heterogeneous distribution of mitochondria and succinate dehydrogenase activity in human airway smooth muscle cells

Postnatal survival of phrenic motor neurons is promoted by BDNF/TrkB.FL signaling

Molecular mechanisms underlying TNFα-induced mitochondrial fragmentation in human airway smooth muscle cells

Chloroquine Affects Presynaptic Membrane Retrieval in Diaphragm Neuromuscular Junctions of Old Mice

Hypercapnia impacts neural drive and timing of diaphragm neuromotor control

Cell-Based Measurement of Mitochondrial Function in Human Airway Smooth Muscle Cells

Size-dependent differences in mitochondrial volume density in phrenic motor neurons

Novel regenerative drug, SPG302 promotes functional recovery of diaphragm muscle activity after cervical spinal cord injury

Electrophysiological effects of BDNF and TrkB signaling at type-identified diaphragm neuromuscular junctions

Molecular Mechanisms Underlying TNFα-Induced Mitochondrial Biogenesis in Human Airway Smooth Muscle

Aging affects the number and morphological heterogeneity of rat phrenic motor neurons and phrenic motor axons

Chloroquine impairs maximal transdiaphragmatic pressure generation in old mice

Neuromotor control of spontaneous quiet breathing in awake rats evaluated by assessments of diaphragm EMG stationarity

Chemical Chaperone 4-PBA Mitigates Tumor Necrosis Factor Alpha-Induced Endoplasmic Reticulum Stress in Human Airway Smooth Muscle

Diaphragm muscle function in a mouse model of early-onset spasticity

Mitochondrial adaptations to inactivity in diaphragm muscle fibers

Effects of arginase inhibition on myocardial Ca 2+ and contractile responses

Primary cilia in satellite cells are the mechanical sensors for muscle hypertrophy

Collaborators (5)

Jin Sun Cho

Carlos Mantilla

Haitao Wang

Kai He

John Halliwill

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