GE PENG

Juntendo University

Japan

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Articles (10)

Year:

2025

Year:

2025

Year:

2024

Year:

2023

Year:

2023

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2023

Year:

2023

Year:

2023

Year:

2023

Year:

2022

Collaborators (4)

Hai Le Thanh Nguyen

Juntendo University

JAPAN

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Masaaki Komatsu

Juntendo University

JAPAN

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Yoshie Umehara

Juntendo University

JAPAN

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François Niyonsaba

Juntendo University Graduate School of Medicine

JAPAN

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GE PENG

Research Interests

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Articles (10)

The interaction between the skin microbiome and antimicrobial peptides within the epidermal immune microenvironment: Bridging insights into atopic dermatitis

AMP-IBP5: A Multifunctional Antimicrobial Peptide for Advanced Wound Healing and Inflammatory Skin Disorders

Calcitriol modulates epidermal tight junction barrier function in human keratinocytes

Cathelicidin LL-37 Activates Human Keratinocyte Autophagy through the P2X₇, Mechanistic Target of Rapamycin, and MAPK Pathways

Calcitriol, an Active Form of Vitamin D3, Mitigates Skin Barrier Dysfunction in Atopic Dermatitis NC/Nga Mice

The Antimicrobial Peptide AMP-IBP5 Suppresses Dermatitis-like Lesions in a Mouse Model of Atopic Dermatitis through the Low-Density Lipoprotein Receptor-Related Protein-1 Receptor

Calcitriol, an Active Form of Vitamin D3, Mitigates Skin Barrier Dysfunction in Atopic Dermatitis NC/Nga Mice

Cathelicidin LL-37 Activates Human Keratinocyte Autophagy through the P2X<sub>7</sub>, Mechanistic Target of Rapamycin, and MAPK Pathways

The Antimicrobial Peptide AMP-IBP5 Suppresses Dermatitis-like Lesions in a Mouse Model of Atopic Dermatitis through the Low-Density Lipoprotein Receptor-Related Protein-1 Receptor

Human-β-defensin-3 attenuates atopic dermatitis-like inflammation through autophagy activation and the aryl hydrocarbon receptor signaling pathway.

Collaborators (4)

Hai Le Thanh Nguyen

Masaaki Komatsu

Yoshie Umehara

François Niyonsaba

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