Geir Slupphaug

Professor

Norwegian University of Science and Technology (NTNU)

Norway

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Geir Slupphaug is a Professor at the Norwegian University of Science and Technology (NTNU), Norway. His research focuses on various aspects of cancer biology, metabolic disorders, and proteomic analysis, as highlighted in recent articles. His work includes studying the inflammatory responses in glioma cells, histidine methylation processes, and the effects of alginate-based microbeads on cellular responses. Additionally, he investigates the implications of monoclonal gammopathy and the mechanisms of DNA repair and methylation.

Articles (14)

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2025

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2025

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2025

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2024

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2021

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2021

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2021

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2020

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2020

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2020

Collaborators (4)

Pål Falnes

University of Oslo

NORWAY

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Finn Lillelund Aachmann

Professor

NORWAY

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Michael O. Hottiger

University of Zurich

SWITZERLAND

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Sebastian Leidel

Full Professor for Cellular RNA Biochemistry

University of Bern

SWITZERLAND

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Geir Slupphaug

Research Interests

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Articles (14)

Identification of substrates and sequence requirements for CARNMT1-mediated histidine methylation of C3H zinc fingers.

Orthologues of the human protein histidine methyltransferase METTL9 display distinct substrate specificities.

Type 1 diabetes mellitus (T1DM) does not affect whole blood responses to alginate-based microspheres despite plasma lipid and glucose differences

Proteomic Analysis Uncovers Enhanced Inflammatory Phenotype and Distinct Metabolic Changes in IDH1 Mutant Glioma Cells

Fucoidan alginate and sulfated alginate microbeads induce distinct coagulation, inflammatory and fibrotic responses

Covalent PARylation of DNA base excision repair proteins regulates DNA demethylation

Progression of monoclonal gammopathy of undetermined significance to multiple myeloma is associated with enhanced translational quality control and overall loss of surface antigens

Proteomic analysis reveals mechanisms underlying increased efficacy of bleomycin by photochemical internalization in bladder cancer cells

RPA2 winged-helix domain facilitates UNG-mediated removal of uracil from ssDNA; implications for repair of mutagenic uracil at the replication fork

Correction to: HDACi mediate UNG2 depletion, dysregulated genomic uracil and altered expression of oncoproteins and tumor suppressors in B- and T-cell lines

Long-Term Exposure to Nanosized TiO2 Triggers Stress Responses and Cell Death Pathways in Pulmonary Epithelial Cells

The human methyltransferase ZCCHC4 catalyses N6-methyladenosine modification of 28S ribosomal RNA.

HDACi mediate UNG2 depletion, dysregulated genomic uracil and altered expression of oncoproteins and tumor suppressors in B- and T-cell lines

RNA in DNA repair

Collaborators (4)

Pål Falnes

Finn Lillelund Aachmann

Michael O. Hottiger

Sebastian Leidel

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